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Causes of hyperthermia include dehydration, use of certain medications, using cocaine and amphetamines or excessive alcohol use. [43] Bodily temperatures greater than 37.5–38.3 °C (99.5–100.9 °F) can be diagnosed as a hyperthermic case. [ 43 ]
In a 2023 study published in the journal Alcohol, chronic heavy alcohol consumption was found to cause DNA damage and oxidative stress, which can lead to an increased risk of cancer ...
The level of ethanol consumption that minimizes the risk of disease, injury, and death is subject to some controversy. [16] Several studies have found a J-shaped relationship between alcohol consumption and health, [17] [18] [2] [19] meaning that risk is minimized at a certain (non-zero) consumption level, and drinking below or above this level increases risk, with the risk level of drinking a ...
Even light to moderate alcohol consumption can have negative effects on health, [8] [9] [10] such as by increasing a person's risk of developing several cancers. [11] [12] A 2014 World Health Organization (WHO) report found that harmful alcohol consumption caused about 3.3 million deaths annually worldwide. [13]
Aragam co-authored a 2022 study that also found a trend of healthy lifestyle habits among light to moderate drinkers, but concluded nonetheless that any level of alcohol consumption increased the ...
May Cause Alcohol Dependence “Regular or excessive alcohol consumption can lead to alcohol-related problems, including addiction, liver damage, and increased risk of certain cancers,” says ...
The diagnosis of alcoholic cardiomyopathy is typically made based on a combination of the patient's medical history, physical examination, and diagnostic tests. [8] Firstly, the doctor will ask the patient about their alcohol consumption habits, as well as any symptoms they may have experienced, such as shortness of breath or swelling in the ...
Alcoholic ketoacidosis is caused by complex physiology that is the result of prolonged and heavy alcohol intake, usually in the setting of poor nutrition. Chronic alcohol use can cause depleted hepatic glycogen stores and ethanol metabolism further impairs gluconeogenesis.