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G6PD converts G6P into 6-phosphoglucono-δ-lactone and is the rate-limiting enzyme of the pentose phosphate pathway. Thus, regulation of G6PD has downstream consequences for the activity of the rest of the pentose phosphate pathway. Glucose-6-phosphate dehydrogenase is stimulated by its substrate G6P.
G6PD deficiency results from mutations in the G6PD gene. G6PD gene contributes to the production of glucose-6-phosphate dehydrogenase. Chemical reactions involving glucose-6-phosphate dehydrogenase produce compounds that prevent reactive oxygen species from building up to toxic levels within red blood cells. If a reduction in the amount of ...
The Beutler test, also known as the fluorescent spot test, [1] is a screening test used to identify enzyme defects. [2] [3] [4] ... "G6PD deficiency". Blood. 84 (11): ...
Hypersensitive response (HR) is a mechanism used by plants to prevent the spread of infection by microbial pathogens.HR is characterized by the rapid death of cells in the local region surrounding an infection and it serves to restrict the growth and spread of pathogens to other parts of the plant.
The nutrient content of a plant can be assessed by testing a sample of tissue from that plant. These tests are important in agriculture since fertilizer application can be fine-tuned if the plants nutrient status is known. Nitrogen most commonly limits plant growth and is the most managed nutrient.
Because the optimal range is different for every plant, gardeners should learn the pH requirements of each one. For example, carnations require a pH range of 6.7-6.9; turfgrasses, 6.3-6.8 ...
A plant line with acceptable resistance against one pathogen may lack resistance against others. Breeding for resistance typically includes: Identification of plants that may be less desirable in other ways, but which carry a useful disease resistance trait, including wild plant lines that often express enhanced resistance.
A plant peptide hormone, systemin, was first identified in tomato plants and genetic modification has been used to demonstrate its function, by adding antisense genes to silence the native gene or by adding extra copies of the native gene. [46] [47]