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Like any drug, taking beta blockers comes with side effects. Churchwell said they can cause bronchial constriction, which can be a problem for people with chronic lung disease, asthma and diabetes.
Bisoprolol, sold under the brand name Zebeta among others, is a beta blocker which is selective for the beta-1 receptor [7] and used for cardiovascular diseases, [7] including tachyarrhythmias, high blood pressure, angina, and heart failure. [7] [8] It is taken by mouth. [7]
Beta blockers vary in their lipophilicity (fat solubility) and in turn in their ability to cross the blood–brain barrier and exert effects in the central nervous system. [76] Beta blockers with greater blood–brain barrier permeability can have both neuropsychiatric therapeutic benefits and side effects, as well as adverse cognitive effects ...
Beta-blockers with intrinsic sympathomimetic activity: acebutolol, pindolol; Some common side effects include increased airway resistance for non-selective beta-blockers, exacerbation of peripheral vascular diseases, and hypotension [15] Beta-blockers are contraindicated in patients with second- or third-degree atrioventricular block.
Beta blockers work by blocking the effects of adrenaline, aka slowing your heart rate and reducing those physical signs and symptoms of nervousness and anxiety, he explained.
While some studies suggest that there are minimal differences in side effects between asthma patients and non-asthma patients, beta 1 blockers are generally avoided in patients with asthma or chronic obstructive pulmonary disease due to their potential to block beta 2 receptors, particularly at high doses.
Atenolol is classified as a beta blocker with low lipophilicity and hence lower potential for crossing the blood–brain barrier and entering the brain. [44] This in turn may result in fewer effects in the central nervous system as well as a lower risk of neuropsychiatric side effects. [44] Only small amounts of atenolol are said to enter the ...
Labetalol is often classified as a beta blocker with low lipophilicity and hence lower potential for crossing the blood–brain barrier and blood–placenta barrier. [17] [29] [30] This in turn may result in fewer effects in the central nervous system as well as a lower risk of neuropsychiatric side effects. [17]