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In men, hyperprolactinemia leads to hypoactive sexual desire and, occasionally, erectile dysfunction. However, the link between erectile dysfunction and prolactin levels are not conclusive. When prolactin levels return to normal in these individuals, sexual desire fully recovers; however, erectile dysfunction only partially recovers. [34]
Sexual desire is not increased in women with polycystic ovary syndrome (PCOS) in spite of high testosterone levels. [28] Women with PCOS actually experience an improvement in sexual desire following treatment of their condition, likely due improved psychological functioning (e.g., body image). [28]
Prolactin (PRL), also known as lactotropin and mammotropin, is a protein best known for its role in enabling mammals to produce milk. It is influential in over 300 separate processes in various vertebrates, including humans. [5] Prolactin is secreted from the pituitary gland in response to eating, mating, estrogen treatment, ovulation and ...
Drugs and sexual desire is about sexual desire being manipulated through drugs from various approaches. Sexual desire is generated under the effects from sex hormones and microcircuits from brain regions. [1] Neurotransmitters play essential roles in stimulating and inhibiting the processes that lead to libido production in both men and women.
This disorder is more common in females than males. Although the tumor itself may not cause symptoms, the tumor can cause an increased production of prolactin and decreased levels of estrogen and testosterone. [3] Symptoms of both sexes could include osteoporosis, infertility, or a decrease in sexual desire. Lactotropic Cells (in blue)
Sexual anhedonia, also known as pleasure dissociative orgasmic disorder, is a condition in which an individual cannot feel pleasure (see anhedonia) from an orgasm. It is thought to be a variant of hypoactive sexual desire disorder .
Prolactin additionally appears to have a direct inhibitory effect on sexual desire in men that is independent of hypogonadism. [68] [69] [70] In men with hyperprolactinemia and hypogonadism, sexual desire is not restored by testosterone replacement therapy, but is restored by prolactin-suppressing medications.
Prolactin inhibits GnRH neurons and therefore inhibits the subsequent release of LH, FSH and sex steroids. The mechanism of prolactin induced inhibition of GnRH release is poorly understood. [13] It is suspected that the prolactin receptor is expressed on a small subset of GnRH neurons in mice and thus has a direct inhibitory effect on GnRH ...