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Ocular hypertension is treated with either medications (eye drops), surgery, or laser. Treatment, by lowering the intraocular pressure, may help decrease the risk of vision loss and damage to the eye from glaucoma. Treatment options include pressure-lowering 'antiglaucomatous' eye drops, surgery, and/or laser eye surgery. [4]
A greyout is a transient loss of vision characterized by a perceived dimming of light and color, sometimes accompanied by a loss of peripheral vision. [1] It is a precursor to fainting or a blackout and is caused by hypoxia (low brain oxygen level), often due to a loss of blood pressure. Greyouts have a variety of possible causes:
Intraocular pressure varies throughout the night and day. The diurnal variation for normal eyes is between 3 and 6 mmHg and the variation may increase in glaucomatous eyes. During the night, intraocular pressure may not decrease [17] despite the slower production of aqueous humour. [18]
Angle-closure glaucoma should be considered if there is painful loss of vision with a red eye, nausea or vomiting. [4] The eye pressure will be very high typically greater than 40 mmHg. [5] Emergent laser treatment to the iris may prevent blindness. [4]
The prognosis for CSR is generally excellent. While immediate vision loss may be as poor as 20/200 in the affected eye, clinically, over 90% of patients regain 20/25 vision or better within 45 days. [1] Once the fluid has resolved, either spontaneously or through treatment, distortion is reduced and visual acuity improves as the eye heals.
Cataract in human eye . Post-Cataract Surgery: After cataract surgery, the natural lens, which filters blue light, is replaced with a synthetic lens that allows more blue light to pass through. This sudden increase in blue light reaching the retina causes the brain to perceive a blue tint in vision.
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Over many years, glaucoma has been defined by an intraocular pressure of more than 20 mm Hg. Incompatible with this (now obsolete) definition of glaucoma was the ever larger number of cases that have been reported in medical literature in the 1980s and 1990s who had the typical signs of glaucomatous damage, like optic nerve head excavation and thinning of the retinal nerve fiber layer, while ...