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A549 cells are adenocarcinomic human alveolar basal epithelial cells, and constitute a cell line that was first developed in 1972 by D. J. Giard, et al. through the removal and culturing of cancerous lung tissue in the explanted tumor of a 58-year-old caucasian male. [1]
This article provides a list of autoimmune diseases. These conditions, where the body's immune system mistakenly attacks its own cells, affect a range of organs and systems within the body. Each disorder is listed with the primary organ or body part that it affects and the associated autoantibodies that are typically found in people diagnosed ...
Diseases and disorders DAS Developmental apraxia of speech: DBA Diamond–Blackfan anemia: DBMD Duchenne–Becker muscular dystrophy DD Developmental disability: DEF Deaf: DF Dengue fever: DH Developmentally handicapped: DHF Dengue hemorrhagic fever: DHF Diastolic heart failure: DHPR Dihydropteridine reductase deficiency: DI Diabetes insipidus: DIC
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The following is a list of genetic disorders and if known, type of mutation and for the chromosome involved. Although the parlance "disease-causing gene" is common, it is the occurrence of an abnormality in the parents that causes the impairment to develop within the child. There are over 6,000 known genetic disorders in humans.
According to Kukushkin, the memories stored in non-brain cells in other parts of the body are memories strictly related to the roles that those specific cells play in human health. Thus, he detailed:
Nevoid basal-cell carcinoma syndrome; Nezelof syndrome; Nicolaides–Baraitser syndrome; Nicolau–Balus syndrome; Night eating syndrome; Nijmegen breakage syndrome; Njølstad syndrome; Nodding disease; Non-24-hour sleep–wake disorder; Noonan syndrome with multiple lentigines; Noonan syndrome; Norman–Roberts syndrome; Northern epilepsy ...
Anderson and colleagues from St Thomas' Hospital, London, were the first to mention a case with possible clinical findings of LEMS in 1953, [11] but Edward H. Lambert, Lee Eaton, and E.D. Rooke at the Mayo Clinic were the first physicians to substantially describe the clinical and electrophysiological findings of the disease in 1956.
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