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Major factors influencing cardiac output – heart rate and stroke volume, both of which are variable. [1]In cardiac physiology, cardiac output (CO), also known as heart output and often denoted by the symbols , ˙, or ˙, [2] is the volumetric flow rate of the heart's pumping output: that is, the volume of blood being pumped by a single ventricle of the heart, per unit time (usually measured ...
Cardiac output as shown on an ECG. Cardiac output (CO) is a measurement of the amount of blood pumped by each ventricle (stroke volume, SV) in one minute. To calculate this, multiply stroke volume (SV), by heart rate (HR), in beats per minute. [1] It can be represented by the equation: CO = HR x SV [1]
Cardiac output (= heart rate * stroke volume. Can also be calculated with Fick principle, palpating method.) Stroke volume (= end-diastolic volume − end-systolic volume) Ejection fraction (= stroke volume / end-diastolic volume) Cardiac output is mathematically ` to systole [clarification needed] Inotropic, chronotropic, and dromotropic states
High-output heart failure is a heart condition that occurs when the cardiac output is higher than normal because of increased peripheral demand. There is a circulatory overload which may lead to pulmonary edema secondary to an elevated diastolic pressure in the left ventricle .
Therefore, using the assumed Fick determination, the approximated cardiac output for an average man (1.9 m3) is: Cardiac Output = (125 mL O 2 /minute × 1.9) / (200 mL O 2 /L − 150 mL O 2 /L) = 4.75 L/min. Cardiac output may also be estimated with the Fick principle using production of carbon dioxide as a marker substance. [3]
Afterload is a determinant of cardiac output. [1] Cardiac output is the product of stroke volume and heart rate. [2] Afterload is a determinant of stroke volume (in addition to preload, and strength of myocardial contraction).
The Frank-Starling mechanism allows the cardiac output to be synchronized with the venous return, arterial blood supply and humoral length, [2] without depending upon external regulation to make alterations. The physiological importance of the mechanism lies mainly in maintaining left and right ventricular output equality.
This allows the heart to cope with the required cardiac output at a relatively low right atrial pressure. We get what is known as a family of cardiac function curves, as the heart rate increases before the plateau is reached, and without the RAP having to rise dramatically to stretch the heart more and get the Starling effect. [citation needed]