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Nitrogen dioxide is an irritant of the mucous membrane linked with another air pollutant that causes pulmonary diseases such as obstructive lung disease, asthma, chronic obstructive pulmonary disease and sometimes acute exacerbation of COPD and in fatal cases, deaths. [2]
There is strong evidence that NO x respiratory exposure can trigger and exacerbate existing asthma symptoms, and may even lead to the development of asthma over longer periods of time. It has also been associated with heart disease, diabetes, birth outcomes, and all-cause mortality, but these nonrespiratory effects are less well-established. [29]
Nitrogen dioxide is poisonous and can be fatal if inhaled in large quantities. [8] Cooking with a gas stove produces nitrogen dioxide which causes poorer indoor air quality. Combustion of gas can lead to increased concentrations of nitrogen dioxide throughout the home environment which is linked to respiratory issues and diseases.
[30] [31] Ground-level ozone, sulfur dioxide, nitrogen dioxide and carbon monoxide are especially harmful for senior citizens, children, and people with heart and lung conditions such as emphysema, bronchitis, and asthma. [14] It can inflame breathing passages, decrease the lungs' working capacity, cause shortness of breath, pain when inhaling ...
[5] [6] It is a significant risk factor for a number of pollution-related diseases, including heart disease, stroke, chronic obstructive pulmonary disease (COPD), asthma and lung cancer. [ 7 ] [ 6 ] It is the fourth-largest risk factor overall for human health [ 8 ] as 99% of people are exposed to harmful levels of air pollution. [ 9 ]
You’re also not alone, as more than 25 million Americans suffer from asthma. This chronic disease causes the lung’s airways to become inflamed and narrowed, making it difficult to breathe.
Asthma phenotyping and endotyping has emerged as a novel approach to asthma classification inspired by precision medicine which separates the clinical presentations of asthma, or asthma phenotypes, from their underlying causes, or asthma endotypes. The best-supported endotypic distinction is the type 2-high/type 2-low distinction.
Occupational asthma is new onset asthma or the recurrence of previously quiescent asthma directly caused by exposure to an agent at workplace. It is an occupational lung disease and a type of work-related asthma. Agents that can induce occupational asthma can be grouped into sensitizers and irritants. [1]