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2. Stroke volume - Wikipedia

   en.wikipedia.org/wiki/Stroke_volume

   In cardiovascular physiology, stroke volume (SV) is the volume of blood pumped from the ventricle per beat. Stroke volume is calculated using measurements of ventricle volumes from an echocardiogram and subtracting the volume of the blood in the ventricle at the end of a beat (called end-systolic volume [note 1]) from the volume of blood just prior to the beat (called end-diastolic volume).

3. Preload (cardiology) - Wikipedia

   en.wikipedia.org/wiki/Preload_(cardiology)

   Heart during ventricular diastole. In cardiac physiology, preload is the amount of sarcomere stretch experienced by cardiac muscle cells, called cardiomyocytes, at the end of ventricular filling during diastole. [1] Preload is directly related to ventricular filling. As the relaxed ventricle fills during diastole, the walls are stretched and ...

4. Cardiac output - Wikipedia

   en.wikipedia.org/wiki/Cardiac_output

   Major factors influencing cardiac output – heart rate and stroke volume, both of which are variable. [1]In cardiac physiology, cardiac output (CO), also known as heart output and often denoted by the symbols , ˙, or ˙, [2] is the volumetric flow rate of the heart's pumping output: that is, the volume of blood being pumped by a single ventricle of the heart, per unit time (usually measured ...

5. Frank–Starling law - Wikipedia

   en.wikipedia.org/wiki/Frank–Starling_law

   The Frank–Starling law of the heart (also known as Starling's law and the Frank–Starling mechanism) represents the relationship between stroke volume and end diastolic volume. [1] The law states that the stroke volume of the heart increases in response to an increase in the volume of blood in the ventricles, before contraction (the end ...

6. Cardiac physiology - Wikipedia

   en.wikipedia.org/wiki/Cardiac_physiology

   Many of the factors that regulate the heart rate also affect cardiac function by altering the stroke volume. While a number of variables are involved, stroke volume is dependent upon the difference between end diastolic volume and end systolic volume. The three primary factors involved are preload, afterload and contractility. [1]

7. End-diastolic volume - Wikipedia

   en.wikipedia.org/wiki/End-diastolic_volume

   End-diastolic volume. In cardiovascular physiology, end-diastolic volume (EDV) is the volume of blood in the right or left ventricle at end of filling in diastole which is amount of blood present in ventricle at the end of diastole. [1] Because greater EDVs cause greater distention of the ventricle, EDV is often used synonymously with preload ...

8. Restrictive cardiomyopathy - Wikipedia

   en.wikipedia.org/wiki/Restrictive_cardiomyopathy

   Specialty. Cardiology. Restrictive cardiomyopathy (RCM) is a form of cardiomyopathy in which the walls of the heart are rigid (but not thickened). [2][3] Thus the heart is restricted from stretching and filling with blood properly. It is the least common of the three original subtypes of cardiomyopathy: hypertrophic, dilated, and restrictive.

9. End-systolic volume - Wikipedia

   en.wikipedia.org/wiki/End-systolic_volume

   End-systolic volume (ESV) is the volume of blood in a ventricle at the end of contraction, or systole, and the beginning of filling, or diastole. ESV is the lowest volume of blood in the ventricle at any point in the cardiac cycle. The main factors that affect the end-systolic volume are afterload and the contractility of the heart.