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Identifying a direct cause for insomnia, particularly in cases of primary insomnia, can be challenging. However, various issues can contribute. Here are some of the most common causes and risk ...
REM sleep deprivation causes an increase in noradrenaline (which incidentally causes the person sleep deprived to be stressed) due to the neurons in the locus coeruleus producing it not ceasing to do so, which causes an increase in the activity of the Na⁺/K⁺-ATPase pump, which itself activates the intrinsic pathway of apoptosis [41] and ...
Insomnia can be short term, lasting for days or weeks, or long term, lasting more than a month. [1] The concept of the word insomnia has two distinct possibilities: insomnia disorder (ID) or insomnia symptoms, and many abstracts of randomized controlled trials and systematic reviews often underreport on which of these two possibilities the word ...
Idiopathic hypersomnia (IH) is a neurological disorder which is characterized primarily by excessive sleep and excessive daytime sleepiness (EDS). [1] Idiopathic hypersomnia was first described by Bedrich Roth in 1976, and it can be divided into two forms: polysymptomatic and monosymptomatic.
In the long term, these cognitive changes may develop into psychiatric disorders and neurodegenerative diseases like Alzheimer's disease. Epigenomic changes such as changes in DNA methylation , histone modifications, and changes in non-coding RNA caused by sleep deprivation may be the cause of these phenotypic presentations.
Cognitive behavioral therapy for insomnia (CBT-I) is a therapy that can help individuals fall asleep faster and stay asleep longer. It is typically a 6- to 8-week treatment plan for long-term ...
Experiments indicated that, in healthy children and adults, episodes of fragmented sleep or insomnia increased sympathetic activation, which can disrupt mood and cognition. The long term consequences include metabolic issues such as glucose homeostasis disruption and even tumor formation and increased risks of cancer.
Insomnia and wake-time sleepiness are related to misalignment between the timing of a non-standard wake–sleep schedule and the endogenous circadian propensity for sleep and wake. In addition to circadian misalignment, attempted sleep at unusual times can be interrupted by noise, social obligations, and other factors.