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  2. Mycotoxins in animal feed - Wikipedia

    en.wikipedia.org/wiki/Mycotoxins_in_animal_feed

    This disease was the turning point for the use of the term mycotoxin. In the 1960s, about 100,000 turkey poults died near London, England due to peanut meal that was contaminated by Mycotoxins produced by Aspergillus flavus. Studies showed that the age group that was most affected was turkeys from two to twenty weeks old.

  3. Wikipedia:WikiProject/NAL/Mycotoxin Detection - Wikipedia

    en.wikipedia.org/.../NAL/Mycotoxin_Detection

    In 2013, she joined the Mycotoxin Prevention and Applied Microbiology Research Team at the National Center for Agricultural Utilization Research in Peoria, IL to pursue research investigating the effects of abiotic stress on crop-fungal pathogen interactions in a manner that influences downstream mycotoxin production and grain contamination.

  4. Gliotoxin - Wikipedia

    en.wikipedia.org/wiki/Gliotoxin

    Gliotoxin is a sulfur-containing mycotoxin that belongs to a class of naturally occurring 2,5-diketopiperazines [1] produced by several species of fungi, especially those of marine origin.

  5. Mycotoxin - Wikipedia

    en.wikipedia.org/wiki/Mycotoxin

    A mycotoxin (from the Greek μύκης mykes, "fungus" and τοξικός toxikos, "poisonous") [1] [2] is a toxic secondary metabolite produced by fungi [3] [4] and is capable of causing disease and death in both humans and other animals.

  6. Aflatoxin - Wikipedia

    en.wikipedia.org/wiki/Aflatoxin

    Prevention [ edit ] A primary means of limiting risk from aflatoxins in the food supply is food hygiene in the commercial commodity supply chain, such as rejecting moldy grain for use in food processing plants and testing of batches of ingredients for aflatoxin levels before adding them to the mix.

  7. Trichothecene - Wikipedia

    en.wikipedia.org/wiki/Trichothecene

    However, if an area has already been contaminated with trichothecene toxins, there are a variety of possible decontamination strategies to prevent further exposure. Treatment with 1% sodium hypochlorite (NaOCl) in 0.1M sodium hydroxide (NaOH) for 4–5 hours has been shown to inhibit the biological activity of T-2 toxin. [ 70 ]

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