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  2. NFATC1 - Wikipedia

    en.wikipedia.org/wiki/NFATC1

    The product of this gene is a component of the nuclear factor of activated T cells DNA-binding transcription complex. This complex consists of at least two components: a preexisting cytosolic component that translocates to the nucleus upon T cell receptor (TCR) stimulation, and an inducible nuclear component. Proteins belonging to this family ...

  3. NFAT - Wikipedia

    en.wikipedia.org/wiki/NFAT

    T-cell receptor (TCR) stimulation causes the dephosphorylation of NFAT which in almost every kind of T cell then forms a complex with AP-1 (except in Tregs). This complex depending on the cytokine context then activates the key transcription factors of the distinct T cell subpopulations: T-bet for Th1, GATA3 for Th2, RORγ for Th17 and BATF for ...

  4. T cell - Wikipedia

    en.wikipedia.org/wiki/T_cell

    CTLA-4 expression is also up-regulated on activated T cells, which in turn outcompetes CD28 for binding to the B7 proteins. This is a checkpoint mechanism to prevent over activation of the T cell. Activated T cells also change their cell surface glycosylation profile. [49] The T cell receptor exists as a complex of several proteins.

  5. CD96 - Wikipedia

    en.wikipedia.org/wiki/CD96

    CD96 (Cluster of Differentiation 96) or Tactile (T cell activation, increased late expression) is a protein that in humans is encoded by the CD96 gene. [5] CD96 is a receptor protein which is expressed on T cells and NK cells and shares sequence similarity with CD226 (also known as DNAM-1). [6]

  6. Autocrine signaling - Wikipedia

    en.wikipedia.org/wiki/Autocrine_signaling

    Autocrine signaling is a form of cell signaling in which a cell secretes a hormone or chemical messenger (called the autocrine agent) that binds to autocrine receptors on that same cell, leading to changes in the cell. [1] This can be contrasted with paracrine signaling, intracrine signaling, or classical endocrine signaling.

  7. Co-stimulation - Wikipedia

    en.wikipedia.org/wiki/Co-stimulation

    Activation of T cells without co-stimulation may lead to the unresponsiveness of the T cell (also called anergy), apoptosis or the acquisition of the immune tolerance. [ 3 ] The counterpart of the co-stimulatory signal is a (co-)inhibitory signal, where inhibitory molecules interact with different signaling pathways in order to arrest T cell ...

  8. NFATC3 - Wikipedia

    en.wikipedia.org/wiki/NFATC3

    The product of this gene is a member of the nuclear factors of activated T cells DNA-binding transcription complex. This complex consists of at least two components: a preexisting cytosolic component that translocates to the nucleus upon T cell receptor (TCR) stimulation and an inducible nuclear component.

  9. Interleukin - Wikipedia

    en.wikipedia.org/wiki/Interleukin

    [15] [16] The protein, which exists in vivo as a monomer, is produced in activated T cells and mast cells, [15] [16] and is activated by the cleavage of an N-terminal signal sequence. [ 16 ] IL3 is produced by T lymphocytes and T-cell lymphomas only after stimulation with antigens, mitogens, or chemical activators such as phorbol esters.