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Type III glycogen storage disease. Can cause less severe hypoglycemia than type I; Phosphoenolpyruvate carboxykinase deficiency, causes metabolic acidosis and severe hypoglycemia. Disorders of fatty acid oxidation; Medium chain acylCoA dehydrogenase deficiency (MCAD) Familial Leucine sensitive hypoglycemia [4]
Inborn errors of metabolism may cause infant hypoglycemia, and much less commonly adult hypoglycemia. [25] Disorders that are related to the breakdown of glycogen, called glycogen storage diseases, may cause hypoglycemia. [3] [25] Normally, breakdown of glycogen leads to increased glucose levels, particularly in a fasting state. [3]
Most neurons have the ability to use other fuels besides glucose (e.g. lactic acid, ketones).Knowledge of the "switchover" process is incomplete. [further explanation needed] The most severe neuroglycopenic symptoms occur with hypoglycemia caused by excess insulin because insulin reduces the availability of other fuels by suppressing ketogenesis and gluconeogenesis.
This state can be either physiologic or pathologic; physiologic ketotic hypoglycemia is a common cause of hypoglycemia in children, often in response to stressors such as infection or fasting. [1] Pathologic ketotic hypoglycemia is typically caused by metabolic defects, such as glycogen storage disorders. [2]
Low blood sugar, or hypoglycemia, can make you feel cold and shaky. “If your body doesn't have enough sugar, it is going to look for ways to try to get more energy and activate things,” says ...
Here is where the concept of "hypoglycemia" as a disorder meets up with all other modern dietary fads such as "natural" foods, vitamin "lack", and "organic" foods. It is also an area in which the hormones are involved. Hence "glandular causes" and "glandular therapy" play a large role. [2]
Idiopathic hypoglycemia is a medical condition in which the glucose level in the blood (blood glucose) is abnormally low due to an undeterminable cause. This is considered an incomplete and unsatisfactory diagnosis by physicians and is rarely used by endocrinologists , as it implies an unfinished diagnostic evaluation.
Medium-chain acyl-CoA dehydrogenase deficiency (MCAD deficiency or MCADD) is a disorder of fatty acid oxidation that impairs the body's ability to break down medium-chain fatty acids into acetyl-CoA. The disorder is characterized by hypoglycemia and sudden death without timely intervention, most often brought on by periods of fasting or vomiting.