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Tachypnea, also spelt tachypnoea, is a respiratory rate greater than normal, resulting in abnormally rapid and shallow breathing. [1]In adult humans at rest, any respiratory rate of 12–20 per minute is considered clinically normal, with tachypnea being any rate above that. [2]
Tachypnea – increased breathing rate; Orthopnea – Breathlessness in lying down position relieved by sitting up or standing; Platypnea – Breathlessness when seated or standing, relieved by lying flat; Trepopnea – Breathlessness when lying flat relieved by lying in a lateral position; Ponopnea – Painful breathing
The respiratory rate in humans is measured by counting the number of breaths for one minute through counting how many times the chest rises. A fibre-optic breath rate sensor can be used for monitoring patients during a magnetic resonance imaging scan. [1] Respiration rates may increase with fever, illness, or other medical conditions. [2]
Shortness of breath (SOB), known as dyspnea (in AmE) or dyspnoea (in BrE), is an uncomfortable feeling of not being able to breathe well enough. The American Thoracic Society defines it as "a subjective experience of breathing discomfort that consists of qualitatively distinct sensations that vary in intensity", and recommends evaluating dyspnea by assessing the intensity of its distinct ...
The time depends on pulse rate, pulmonary function, RBC count, and other metabolic factors. Lidocaine can be given in 1.5 mg/kg IV a few minutes before sedation and paralysis. The purpose of administering lidocaine is to blunt the sympathetic response of an increased heart rate, blood pressure, and intracranial pressure caused by laryngoscopy.
Trepopnea /tɹɛpəʊpˈniːə/ is dyspnea (shortness of breath) that is sensed while lying on one side but not on the other [1] (lateral recumbent position). It results from disease of one lung, one major bronchus, or chronic congestive heart failure that affects only a side of breathing.
Respiratory failure is classified as either Type 1 or Type 2, based on whether there is a high carbon dioxide level, and can be acute or chronic. In clinical trials, the definition of respiratory failure usually includes increased respiratory rate, abnormal blood gases (hypoxemia, hypercapnia, or both), and evidence of increased work of breathing.
Causes may include heart failure, kidney failure, narcotic poisoning, intracranial pressure, and hypoperfusion of the brain (particularly of the respiratory center). The pathophysiology of Cheyne–Stokes breathing can be summarized as apnea leading to increased CO 2 which causes excessive compensatory hyperventilation, in turn causing decreased CO 2 which causes apnea, restarting the cycle.