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Trigeminal neuralgia (TN or TGN), also called Fothergill disease, tic douloureux, trifacial neuralgia, or suicide disease, is a long-term pain disorder that affects the trigeminal nerve, [7] [1] the nerve responsible for sensation in the face and motor functions such as biting and chewing.
Scintillating scotomas are most commonly caused by cortical spreading depression, a pattern of changes in the behavior of nerves in the brain during a migraine. Migraines, in turn, may be caused by genetic influences and hormones. People with migraines often self-report triggers for migraines involving stress or foods, [9] or bright lights. [10]
However, the migraine aura can manifest itself in isolation, that is, without being followed by headache. The aura can stay for the duration of the migraine; depending on the type of aura, it can leave the person disoriented and confused. It is common for people with migraines to experience more than one type of aura during the migraine.
Bleeding into the eye; Temporal arteritis: Inflammation of an artery in the brain that supplies blood to the optic nerve. Migraine headaches: Spots of light, halos, or zigzag patterns are common symptoms prior to the start of the headache. A retinal migraine is when you have only visual symptoms without a headache.
Other sensory experiences, including tingling, numbness, difficulty speaking, or temporary alterations in hearing or smell, may also occur during the aura phase. The pathophysiology of migraines with aura is multifaceted, involving a complex interplay of genetic predispositions, environmental influences, and intricate neurobiological mechanisms.
Percent of women and men who have experienced migraine with or without aura within the last 3 months. Migraine is common, with around 33% of women and 18% of men affected at some point in their lifetime. [142] Onset can be at any age, but prevalence rises sharply around puberty, and remains high until declining after age 50. [142]
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