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Specific language impairment (SLI) (the term developmental language disorder is preferred by some) [1] is diagnosed when a child's language does not develop normally and the difficulties cannot be accounted for by generally slow development, physical abnormality of the speech apparatus, autism spectrum disorder, apraxia, acquired brain damage or hearing loss.
A larger body of research exists around neuroscientific studies with children diagnosed with a specific language impairment (SLI). fMRI studies have shown that children with SLI have a significantly smaller left hemispheric pars triangularis (Broca's area) and asymmetry of dominance of language structures, as opposed to the more typical left ...
Specific language impairment (SLI) is a developmental language disorder among children that has no known cause and cannot be attributed to any physical or intellectual disability, environmental factors such as deprivation, hearing loss, or any other underlying etiology. [15]
Similarly, "specific language impairment" (SLI), commonly used in North America, was considered too narrow as it only focused on language issues without considering other potential difficulties children may face.
Some speech and language impairments have environmental causes. A specific language impairment, for example, may be caused by insufficient language stimulation in the environment. If a child does not have access to an adequate role model, or is not spoken to with much frequency, the child may not develop strong language skills.
Laurence Baker Leonard is an American speech pathologist and linguist, recognized for his research on Specific Language Impairment (SLI) in children. As a leading scholar in this field, he has been selected to receive the biennial Callier Prize in Communication Disorders. [1]
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Specific Language Impairment (SLI) is a language impairment in which a child’s language develops atypically, but their difficulties cannot be attributed to factors such as autism or slow development. Curtiss's study aimed to determine if there was a probable genetic factor involved in SLI.