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The classification of central polydactyly is based on the extent of duplication and involves the following three types: Type I is a central duplication, not attached to the adjacent finger by osseous or ligamentous attachments; it frequently does not include bones, joints, cartilage, or tendons.
Polydactyly There are probably many genes, both dominant and recessive, that cause polydactyly in cats. Most cases of polydactyly in cats are perfectly harmless. Pd Thumb-cat polydactyly gene. The Pd gene (dominant with incomplete penetrance) causes the benign, pre-axial form of polydactyly where one or more extra toes occur near the dew claw.
One of the polydactyl cats at the Ernest Hemingway House in Key West, Florida.This particular cat has seven (two extra) toes on each paw. A polydactyl cat is a cat with a congenital physical anomaly called polydactyly (also known as polydactylism or hyperdactyly), which causes the cat to be born with more than the usual number of toes on one or more of its paws.
Synpolydactyly is a combination of syndactyly and polydactyly. This image shows the hand morphology of an individual with polydactyly. SPD is inherited in an autosomal dominant pattern, meaning that an individual only needs to inherit one copy of the affected gene, also known as an allele, from either parent to potentially develop the condition.
The term, however, has been found offensive by some because of the reference to the polydactyly, which is seen as a 'freakish' label. [citation needed] For this reason, six-fingered dwarfism has been removed as an alternative name for this entry. This leaves Ellis–Van Creveld syndrome with its initialism, EVC, as the only satisfactory ...
Inheritance of abnormal genes, e.g. polydactyly, ectrodactyly or brachydactyly, symptoms of deformed limbs then often occur in combination with other symptoms ; external causes during pregnancy (thus not inherited), e.g. via amniotic band syndrome; teratogenic drugs (e.g. thalidomide, which causes phocomelia) or environmental chemicals
Absent tibia-polydactyly-arachnoid cyst syndrome, also known as Holmes-Collins syndrome, is a very rare multi-systemic hereditary disorder which is characterized by facial dysmorphisms, [1] pre/post-axial polydactyly, toe syndactyly, missing/underdeveloped tibia bone, and the presence of a retrocerebellar arachnoid cyst.
The disease is not easily definable. The main form of diagnosis is presumptive, if the person has the usual triad of preaxial polydactyly with cutaneous syndactyly of at least one limb, macrocephaly, and hypertelorism. However, a definitive diagnosis can be made if there is a phenotype that is caused by a Gcps and a Gli3 gene mutation. It can ...