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Bell's palsy is a type of facial paralysis that results in a temporary inability to control the facial muscles on the affected side of the face. [1] In most cases, the weakness is temporary and significantly improves over weeks. [4] Symptoms can vary from mild to severe. [1]
A dosage of up to 200–250 mg daily for a sustained period of six to twelve months may be needed to cure hyperacusis. A possible mechanism of action of this drug is that clomipramine reduces reactions of the autonomic nervous system to sounds. [53] The drug ambroxol helps relieve the pain experienced by several pain hyperacusis patients.
The score predicts recovery in those with Bell's palsy. [2] The score carries the name of the Dr John W. House and Dr Derald E. Brackmann, otolaryngologists in Los Angeles, California, who first described the system in 1985. [1] It is one of a number of facial nerve scoring systems, such as Burres-Fisch, Nottingham, Sunnybrook, [3] and ...
What Bell’s palsy advocates want you to know Azizzadeh notes that Bell’s palsy can be very “socially stressful” for individuals experiencing it, making it important for someone like Gravel ...
Bell's palsy is the most common cause of acute facial nerve paralysis. [3] [4] There is no known cause of Bell's palsy, [5] [6] although it has been associated with herpes simplex infection. Bell's palsy may develop over several days, and may last several months, in the majority of cases recovering spontaneously.
Bell's Mania is a syndrome with unexplained etiology which was first explained by American psychiatrist Luther Bell in the 1850s after observing institutionalized psychiatric patients. The first clinical reports and descriptions of people with acute exhaustive mania and delirium were provided by a few psychiatrists in the United States of ...
In contrast, pseudobulbar palsy is a clinical syndrome similar to bulbar palsy but in which the damage is located in upper motor neurons of the corticobulbar tracts in the mid-pons (i.e., in the cranial nerves IX-XII), that is the nerve cells coming down from the cerebral cortex innervating the motor nuclei in the medulla.
Pseudobulbar palsy is the result of damage of motor fibers traveling from the cerebral cortex to the lower brain stem. This damage might arise in the course of a variety of neurological conditions that involve demyelination and bilateral corticobulbar lesions. Examples include: [3] Progressive supranuclear palsy; Amyotrophic lateral sclerosis