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Although both vasopressors, vasopressin and epinephrine differ in that vasopressin does not have direct effects on cardiac contractility as epinephrine does. [18] Thus, vasopressin is theorized to be of increased benefit over epinephrine in cardiac arrest due to its properties of not increasing myocardial and cerebral oxygen demands. [18]
Tricyclics have a narrow therapeutic index, i.e., the therapeutic dose is close to the toxic dose. [7] Factors that increase the risk of toxicity include advancing age, cardiac status, and concomitant use of other drugs. [8] However, serum drug levels are not useful for evaluating risk of arrhythmia or seizure in tricyclic overdose. [9]
551 11998 Ensembl ENSG00000101200 ENSMUSG00000037727 UniProt P01185 P35455 RefSeq (mRNA) NM_000490 NM_009732 RefSeq (protein) NP_000481 NP_033862 Location (UCSC) Chr 20: 3.08 – 3.08 Mb Chr 2: 130.42 – 130.42 Mb PubMed search Wikidata View/Edit Human View/Edit Mouse Human vasopressin, also called antidiuretic hormone (ADH), arginine vasopressin (AVP) or argipressin, is a hormone synthesized ...
Cardiac arrest (also known as sudden cardiac arrest [SCA] [11]) is when the heart suddenly and unexpectedly stops beating. [ 12 ] [ 1 ] When the heart stops beating, blood cannot properly circulate around the body and the blood flow to the brain and other organs is decreased.
The second goal, is to perfuse the heart itself. Perfusion of the heart is necessary for successful defibrillation (if the arrest type is shockable) and ROSC. [2] This is accomplished during the relaxation phase of CPR as it creates diastole-like conditions. [3] During cardiac arrest, CPP is one of the most important variables associated with ...
(Defibrillation uses a therapeutic dose of electric current to the heart at a random moment in the cardiac cycle, and is the most effective resuscitation measure for cardiac arrest associated with ventricular fibrillation and pulseless ventricular tachycardia. [1])
According to the CDC, the specific objectives of the project are: [7]. To quantify the incidence and location of cardiac arrest events; To track the performance of each component of the Emergency Medical Services system (e.g., 9-1-1 dispatching and pre-arrival phone instructions, bystander care, first responder, ALS ambulance and definitive care)
The well known antidiuretic effect of vasopressin occurs via activation of V 2 R. [1] Vasopressin regulates water excretion from the kidney by increasing the osmotic water permeability of the renal collecting duct – an effect that is explained by coupling of the V 2 R with the G s signaling pathway, which activates cAMP.