Search results
Results from the WOW.Com Content Network
Scleritis can be classified as anterior scleritis and posterior scleritis. Anterior scleritis is the most common variety, accounting for about 98% of the cases. It is of two types : Non-necrotising and necrotising. Non-necrotising scleritis is the most common, and is further classified into diffuse and nodular type based on morphology.
Episcleritis is a benign, self-limiting inflammatory disease affecting part of the eye called the episclera.The episclera is a thin layer of tissue that lies between the conjunctiva and the connective tissue layer that forms the white of the eye ().
If there is an underlying cause, treatment should be given based on the disease. Non specific treatment measures include cycloplegics , corticosteroids and immunosuppressive drugs . [ 2 ] The biologic drugs that are currently used in treatment of panuveitis include anti tumor necrosis factor , cytokine receptor antibodies and interferon-α .
Uveitis is described anatomically, by the part of the eye affected, as anterior, intermediate or posterior, or panuveitic if all parts are involved. Anterior uveitis ( iridocyclitis ) is the most common, with the incidence of uveitis overall affecting approximately 1:4500, most commonly those between the ages of 20–60.
Hyphaema – showing blood filling the anterior chamber, causing a horizontal fluid level. Particular signs and symptoms may indicate that the cause is serious and requires immediate attention. [1] Seven such signs are: Reduced visual acuity; Ciliary flush (circumcorneal injection) Corneal abnormalities including edema or opacities ("corneal haze")
Posterior synechia can be observed in cases of anterior uveitis secondary to severe to moderate bacterial keratitis. [2] Posterior synechia also cause glaucoma, but with a different mechanism. In posterior synechia, the iris adheres to the lens, blocking the flow of aqueous humor from the posterior chamber to the anterior chamber.
Scleroderma is a group of autoimmune diseases that may result in changes to the skin, blood vessels, muscles, and internal organs. [2] [6] [8] The disease can be either localized to the skin or involve other organs, as well. [2]
Treatment of atrophic rhinitis can be either medical or surgical. Medical measures include: Nasal irrigation using normal saline [4] Nasal irrigation and removal of crusts using alkaline nasal solutions. 25% glucose in glycerine can be applied to the nasal mucosa to inhibit the growth of proteolytic organisms which produce foul smell.