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Hypermagnesemia is an electrolyte disorder in which there is a high level of magnesium in the blood. [3] Symptoms include weakness, confusion, decreased breathing rate, and decreased reflexes. Hypermagnesemia can greatly increase the chances of adverse cardiovascular events. [1] [3] Complications may include low blood pressure and cardiac ...
Hypermagnesemia typically occurs in individuals with abnormal kidney function. This imbalance can also occur with use of antacids or laxatives that contain magnesium. Iatrogenic cases of hypermagnesemia can be prevented by avoiding magnesium-containing medications. [citation needed]
Due to magnesium's mobile nature, the plant will first break down chlorophyll in older leaves and transport the Mg to younger leaves which have greater photosynthetic needs. Therefore, the first sign of magnesium deficiency is the chlorosis of old leaves which progresses to the young leaves as the deficiency progresses. [ 57 ]
As a bronchodilator after beta-agonist and anticholinergic agents have been tried, e.g. in severe exacerbations of asthma. [4]Obstetrics: Magnesium sulfate is used to prevent seizures in women with preeclampsia and eclampsia, and is also used for fetal neuroprotection in preterm deliveries, but has been shown to be an ineffective tocolytic agent.
Hyperphosphatemia is a condition characterized by elevated levels of phosphate in the blood, often caused by kidney dysfunction or excessive intake.
Magnesium bromide is used as a mild sedative (this action is due to the bromide, not the magnesium). Magnesium stearate is a slightly flammable white powder with lubricating properties. In pharmaceutical technology, it is used in pharmacological manufacture to prevent tablets from sticking to the equipment while compressing the ingredients into ...
An affidavit previously obtained by the local news stations stated that Jacob left his girlfriend's house, saying he was going to have dinner with his family.
The effect is an electrolyte imbalance similar to that seen with thiazide diuretic therapy (which causes pharmacological inhibition of NCC activity). [ 4 ] Gitelman syndrome was formerly considered a subset of Bartter syndrome until the distinct genetic and molecular bases of these disorders were identified.