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The enamel on primary teeth has a more opaque crystalline form and thus appears whiter than on permanent teeth. The large amount of mineral in enamel accounts not only for its strength but also for its brittleness. [6] Tooth enamel ranks 5 on Mohs hardness scale (between steel and titanium) and has a Young's modulus of 83 GPa. [4]
The AMELX gene encodes for the structural modeling protein, amelogenin, which works with other amelogenesis-related proteins to direct the mineralisation of enamel. This process involves the organization of enamel rods , the basic unit of tooth enamel, as well as the inclusion and growth of hydroxyapatite crystals.
The teeth of Arctodus pristinus transition between undulating to acute-angled Hunter-Schreger bands while Arctodus simus exhibited a transition between undulating to zigzag bands, demonstrating an evolution towards reinforced tooth enamel. This has been convergently evolved with giant pandas, agriotheriin bears, and Hemicyon. [9]
The enamel on primary teeth has a more opaque crystalline form and thus appears whiter than on permanent teeth. The large amount of mineral in enamel accounts not only for its strength but also for its brittleness. [7] Tooth enamel ranks 5 on Mohs hardness scale and has a Young's modulus of 83 GPa. [5]
Amelogenesis is the process of forming tooth enamel, the hard, protective outer layer of teeth. [1] This process begins during tooth development after the initial formation of dentin (dentinogenesis), the layer beneath the enamel. [2]
Enamel and dentin do not regenerate after they mineralize initially. Enamel hypoplasia is a condition in which the amount of enamel formed is inadequate. [59] This results either in pits and grooves in areas of the tooth or in widespread absence of enamel. Diffuse opacities of enamel does not affect the amount of enamel but changes its appearance.
Amalgam filling on first molar. In dentistry, amalgam is an alloy of mercury used to fill teeth cavities. [1] It is made by mixing a combination of liquid mercury and particles of solid metals such as silver, copper or tin.
When follistatin, a BMP inhibitor, is over expressed in the epithelium of developing teeth, the ameloblasts do not differentiate and no enamel forms. Another example includes the conditional deletion of dicer-1 in the epithelium of developing teeth, which may cause impaired differentiation of ameloblasts resulting in deficient enamel formation. [2]