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Basidiomycota that reproduce asexually (discussed below) can typically be recognized as members of this division by gross similarity to others, by the formation of a distinctive anatomical feature (the clamp connection), cell wall components, and definitively by phylogenetic molecular analysis of DNA sequence data.
It is considered to be part of the skin's normal human microbiota [2] [6] and begins to colonize the skin of humans shortly after birth. [7] Malassezia sympodialis, often has a symbiotic or commensal relationship with its host, but it can act as a pathogen causing a number of different skin diseases, [8] such as atopic dermatitis. [8] [9]
Malassezia pachydermatis is a zoophilic yeast in the division Basidiomycota.It was first isolated in 1925 by Fred Weidman, and it was named pachydermatis (Greek for 'thick-skin') after the original sample taken from an Indian rhinoceros (Rhinocerosus unicornis) with severe exfoliative dermatitis. [2]
Identification of Malassezia on skin has been aided by the application of molecular or DNA-based techniques. These investigations show that the M. globosa is the species that causes most skin disease in humans, and that it is the most common cause of dandruff and seborrhoeic dermatitis (though M. restricta is also involved).
Basidiomycota — a Fungus phylum, and a fungi classification group that includes mushrooms Subcategories. This category has the following 9 subcategories, out of 9 ...
The skin rash of tinea versicolor (pityriasis versicolor) is also due to infection by this fungus. As the fungus requires fat to grow, [3] it is most common in areas with many sebaceous glands: on the scalp, [4] face, and upper part of the body. When the fungus grows too rapidly, the natural renewal of cells is disturbed, and dandruff appears ...
Malassezia furfur is a fungus that lives on the superficial layers of the dermis.It generally exists as a commensal organism forming a natural part of the human skin microbiota, but it can gain pathogenic capabilities when morphing from a yeast to a hyphal form during its life cycle, through unknown molecular changes. [2]
The disease results from direct entry of the fungus through broken skin such as an insect bite or trauma, or eating contaminated food. [1] [3] It generally affects people who are well. [2] Diagnosis is by medical imaging, biopsy, microscopy, culture and histopathology. [2] Treatment usually involves amphotericin B and surgery. [3] [4]