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Multi-infarct dementia results from a series of small strokes affecting several brain regions. Stroke-related dementia involving successive small strokes causes a more gradual decline in cognition. [4] Dementia may occur when neurodegenerative and cerebrovascular pathologies are mixed, as in susceptible elderly people (75 years and older).
Binswanger's disease, also known as subcortical leukoencephalopathy and subcortical arteriosclerotic encephalopathy, [1] is a form of small-vessel vascular dementia caused by damage to the white brain matter. [2] White matter atrophy can be caused by many circumstances including chronic hypertension as well as old age. [3]
AFib, the most common heart rhythm disorder, is linked to a 39% increased risk of cognitive impairment, while coronary heart disease increases the risk of dementia by 27%, with up to 50% of heart ...
Well, both AFib and dementia have risk factors in common, including high blood pressure, type 2 diabetes, heart failure, smoking, vascular disease, obstructive sleep apnea and advanced age.
Coronary heart disease is responsible for a 27% higher risk of developing dementia, and after a heart attack, up to 50% of patients may experience cognitive decline.
It is one of the most common causes of disability among the elderly but can develop before the age of 65 when it is known as early-onset dementia or presenile dementia. [ 283 ] [ 284 ] Less than 1% of those with Alzheimer's have gene mutations that cause a much earlier development of the disease, around the age of 45, known as early-onset ...
Inferior left ventricle wall scar, short axis echocardiography view Myocardial scarring is the accumulation of fibrous tissue resulting after some form of trauma to the cardiac tissue. [ 1 ] [ 2 ] Fibrosis is the formation of excess tissue in replacement of necrotic or extensively damaged tissue.
Patients on the blood-thinning drug Warfarin experienced higher rates of dementia and Alzheimer's, according to a new study. Widely-used heart drug linked to increased dementia risk Skip to main ...
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