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Temporal lobe epilepsy is the most common type of focal onset epilepsy among adults. [1] Seizure symptoms and behavior distinguish seizures arising from the mesial (medial) temporal lobe from seizures arising from the lateral (neocortical) temporal lobe. [2] Memory and psychiatric comorbidities may occur.
Alternatively, autobiographical memory loss may result from subtle changes in the temporal lobe which gives rise to temporal lobe epilepsy and to memory problems. The mechanism and etiology of this phenomenon remain controversial, especially as it is impossible to rule out prior subclinical epileptic activity which could be responsible for a ...
Temporal lobe signs usually involve auditory sensation and memory, and may include: [citation needed] deafness without damage to the structures of the ear, described as cortical deafness; tinnitus, auditory hallucinations; loss of ability to comprehend music or language, described as a sensory aphasia (Wernicke's aphasia)
For autobiographical memory, the hippocampus, located in the medial temporal lobe, is involved in the encoding of declarative memory (memory for facts and events), while the temporal cortex is involved in the storage of such memory. [21]
An overlap between symptoms can occur as the disease progresses and spreads through the brain regions. [14] Structural MRI scans often reveal frontal lobe and/or anterior temporal lobe atrophy, but in early cases the scan may seem normal. Atrophy can be either bilateral or asymmetric. [13]
SD is a clinically defined syndrome but is associated with predominantly temporal lobe atrophy (left greater than right) and hence is sometimes called temporal variant FTLD (tvFTLD). [7] SD is one of the three variants of primary progressive aphasia (PPA), which results from neurodegenerative disorders such as FTLD or Alzheimer's disease. There ...
A person experiencing TGA has memory impairment; with an inability to remember events or people from the past few minutes, hours or days (retrograde amnesia) and has working memory of only the past few minutes or less, thus they cannot retain new information or form new memories beyond that period of time (anterograde amnesia). [4]
fMRI scans following brief sleep deprivation (24 hours or less) show increases in thalamic activation. Verbal working memory tasks normally cause increases in left temporal lobe activity. After 35 hours come noted decreases in temporal lobe activation and increases in parietal lobe activation. [42] Working memory model.