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Acquired idiopathic generalized anhidrosis appears to have a variety of etiologies. Theoretically, dysfunction or degeneration of cholinergic sympathetic nerve fibers involved in sweating (sudomotor neuropathy), dysfunction of acetylcholine receptors and/or cholinergic signals (idiopathic pure sudomotor failure may fall under this category), and primary failures of the sweat glands with ...
The LD50 is when half the dogs die at that dose, so the toxic dose may be a lot lower, and if your dog has a health problem or is taking another medication, it may be even lower than that.
The treatment options for hypohidrosis and anhidrosis are largely limited to preventing overheating, and attempting to resolve or prevent further deterioration of any known underlying causes. Those with hypohidrosis should avoid drugs that can aggravate the condition (see "Medications", under § Causes). They should limit activities that raise ...
Idiopathic pure sudomotor failure (IPSF) is the most common cause of a rare disorder known as acquired idiopathic generalized anhidrosis (AIGA), a clinical syndrome characterized by generalized decrease or absence of sweating without other autonomic and somatic nervous dysfunctions and without persistent organic cutaneous lesions.
For these reasons, some psychiatrists are hesitant to prescribe antidepressants for the treatment of bipolar disorder unless mood stabilizers have failed to have an effect, however, others feel that antidepressants still have an important role to play in treatment of bipolar disorder. Side effects vary greatly among different classes of ...
The drug seems ineffective in the treatment of current rapid-cycling, acute mania, or acute depression in bipolar disorder. [25] Lamotrigine has been shown to be as effective as lithium, the standard treatment for bipolar disorder. [26] Lamotrigine has not demonstrated clear efficacy in treating acute mood episodes, either mania or depression.
It is also believed that torsion (twisting) of the thoracic spine can cause blockage of the anterior radicular artery leading to Harlequin syndrome. [8] The sympathetic deficit on the denervated side causes anhidrosis (lack of sweating). Patients with Horner's Syndrome may also experience this.
The second cause (and least common out of the 2) is a genetic mutation in the NFKBIA gene, located in chromosome 14 at the q13.2 locus. [ 14 ] The protein that is encoded by this gene inhibits the NF-κB transcription factor through the masking of nuclear localization signals from NF-κB transcription factor proteins, this process keeps them in ...
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