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Previous research has suggested cafestol might increase cholesterol, particularly low-density lipoprotein (LDL) cholesterol. However, in this study, cafestol did not change LDL, total cholesterol ...
Cafestol may act as an agonist ligand for the nuclear receptor farnesoid X receptor and pregnane X receptor, blocking cholesterol homeostasis. Thus cafestol can increase cholesterol synthesis. [6] Cafestol has also shown anticarcinogenic properties in rats. [7] Cafestol also has neuroprotective effects in a Drosophila fruit fly model of ...
The 2021 European Society of Cardiology Guidelines on Cardiovascular Disease Prevention in Clinical Practice state: "Non-filtered coffee contains LDL-C-raising cafestol and kahweol, and may be associated with an up to 25% increased risk of atherosclerosis (ASCVD) mortality by consumption of nine or more drinks a day.
1071 n/a Ensembl ENSG00000087237 n/a UniProt P11597 n/a RefSeq (mRNA) NM_000078 NM_001286085 n/a RefSeq (protein) NP_000069 NP_001273014 n/a Location (UCSC) Chr 16: 56.96 – 56.98 Mb n/a PubMed search n/a Wikidata View/Edit Human Cholesteryl ester transfer protein (CETP), also called plasma lipid transfer protein, is a plasma protein that facilitates the transport of cholesteryl esters and ...
A new study has identified a protein that helps drive bladder cancer by triggering the synthesis of cholesterol via mouse and cell models. Researchers found that a combination therapy of two drugs ...
Cholesterol is the principal sterol of all higher animals, distributed in body tissues, especially the brain and spinal cord, and in animal fats and oils. [3] [4]Cholesterol is biosynthesized by all animal cells [citation needed] and is an essential structural and signaling component of animal cell membranes.
22R-Hydroxycholesterol, or (3β)-cholest-5-ene-3,22-diol is an endogenous, metabolic intermediate in the biosynthesis of the steroid hormones from cholesterol. [1] [2] Cholesterol ((3β)-cholest-5-en-3-ol) is hydroxylated by cholesterol side-chain cleavage enzyme (P450scc) to form 22R-hydroxycholesterol, which is subsequently hydroxylated again by P450scc to form 20α,22R-dihydroxycholesterol ...
The regulate the metabolic pathway for the elimination of cholesterol. However, there is no physiological ligands was identified. [ 2 ] Xenobiotic receptors recognize the foreign chemicals and trigger detoxification and metabolism pathways in different host tissues.
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