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Some antidepressants, including commonly prescribed ones from the class known as SSRIs (selective serotonin reuptake inhibitors), can cause sexual side effects such as erectile dysfunction ...
On the other hand, significantly more patients drop off from the antidepressant treatment than from psychotherapy, likely because of the side effects of antidepressants. [106] Successful psychotherapy appears to prevent the recurrence of depression even after it has been terminated or replaced by occasional "booster" sessions.
They can kill your libido, make orgasm impossible, and may cause a host of other side effects including weight gain, dry mouth, and diarrhea. For all these reasons, it’s critical to find drugs ...
In addition to post-treatment relapse, depressive symptoms can even recur in the course of long-term therapy (tachyphylaxis). Also, currently available antidepressants all elicit undesirable side-effects, and new agents should be divested of the distressing side-effects of both first and second-generation antidepressants. [6]
Side effects include nervousness, jitteriness, vomiting and tachycardia (increased heart rate). And we all know what happens when you have caffeine too close to bedtime… hello , sleepless night.
Data also shows that using pro-inflammatory agents in the treatment of diseases like melanoma can lead to depression. Several meta-analytical studies have found increased levels of proinflammatory cytokines and chemokines in depressed patients. [190] This link has led scientists to investigate the effects of antidepressants on the immune system.
Antidepressants can cause various adverse effects, depending on the individual and the drug in question. [ 123 ] Almost any medication involved with serotonin regulation has the potential to cause serotonin toxicity (also known as serotonin syndrome ) – an excess of serotonin that can induce mania, restlessness, agitation, emotional lability ...
The pharmacology of antidepressants is not entirely clear.. The earliest and probably most widely accepted scientific theory of antidepressant action is the monoamine hypothesis (which can be traced back to the 1950s), which states that depression is due to an imbalance (most often a deficiency) of the monoamine neurotransmitters (namely serotonin, norepinephrine and dopamine). [1]
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