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  2. KEAP1 - Wikipedia

    en.wikipedia.org/wiki/Keap1

    A series of synthetic oleane triterpenoid compounds, known as antioxidant inflammation modulators (AIMs), are being developed by Reata Pharmaceuticals, Inc. and are potent inducers of the Keap1-Nrf2 pathway, blocking Keap1-dependent Nrf2 ubiquitination and leading to the stabilization and nuclear translocation of Nrf2 and subsequent induction ...

  3. Glycine receptor - Wikipedia

    en.wikipedia.org/wiki/Glycine_receptor

    The glycine receptor (abbreviated as GlyR or GLR) is the receptor of the amino acid neurotransmitter glycine. GlyR is an ionotropic receptor that produces its effects through chloride currents .

  4. Glycine - Wikipedia

    en.wikipedia.org/wiki/Glycine

    Glycine is thought to be a hepatic detoxifier of a number endogenous and xenobiotic organic acids. [44] Bile acids are normally conjugated to glycine in order to increase their solubility in water. [45] The human body rapidly clears sodium benzoate by combining it with glycine to form hippuric acid which is then excreted. [46]

  5. Glycinergic - Wikipedia

    en.wikipedia.org/wiki/Glycinergic

    A glycinergic agent (or drug) is a chemical which functions to directly modulate the glycine system in the body or brain. Examples include glycine receptor agonists , glycine receptor antagonists , and glycine reuptake inhibitors .

  6. Sodium- and chloride-dependent glycine transporter 1 - Wikipedia

    en.wikipedia.org/wiki/Sodium-_and_chloride...

    Elevation of extracellular synaptic glycine concentration by blockade of GlyT1 has been hypothesized to potentiate NMDA receptor function in vivo and to represent a rational approach for the treatment of schizophrenia and cognitive disorders. Several drug candidates have reached clinical trials.

  7. NMDA receptor - Wikipedia

    en.wikipedia.org/wiki/NMDA_receptor

    Memantine is the first drug approved for treatment of severe and more advanced Alzheimer's disease, which for example anticholinergic drugs do not do much good for. [114] It helps recovery of synaptic function and in that way improves impaired memory and learning. [ 19 ]

  8. Glycine cleavage system - Wikipedia

    en.wikipedia.org/wiki/Glycine_cleavage_system

    Glycine encephalopathy, also known as non-ketotic hyperglycinemia (NKH), is a primary disorder of the glycine cleavage system, resulting from lowered function of the glycine cleavage system causing increased levels of glycine in body fluids. The disease was first clinically linked to the glycine cleavage system in 1969. [10]

  9. Glycine transporter - Wikipedia

    en.wikipedia.org/wiki/Glycine_transporter

    Glycine transporters (GlyTs) are plasmalemmal neurotransmitter transporters. They serve to terminate the signaling of glycine by mediating its reuptake from the synaptic cleft back into the presynaptic neurons .

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