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Prelingual hearing loss can be considered congenital, present at birth, or acquired, occurring after birth before the age of one. Congenital hearing loss can be a result of maternal factors (rubella, cytomegalovirus, or herpes simplex virus, syphilis, diabetes), infections, toxicity (pharmaceutical drugs, alcohol, other drugs), asphyxia, trauma, low birth weight, prematurity, jaundice, and ...
A main focus of early intervention programs and services for deaf and hard of hearing children is language development. Early interventionists are able to work with the family during the early, critical years for language acquisition. [62] Early intervention can take many forms and usually depends on where the family lives.
Hearing loss due to chemicals starts in the high-frequency range and is irreversible. It damages the cochlea with lesions and degrades central portions of the auditory system. [45] For some ototoxic chemical exposures, particularly styrene, [46] the risk of hearing loss can be higher than being exposed to noise alone.
Hearing loss is typically gradual and can be difficult to notice at first, but early signs include difficulty hearing in loud or crowded places, when speaking on the phone, or when someone isn’t ...
Hearing loss due to chemicals starts in the high frequency range and is irreversible. It damages the cochlea with lesions and degrades central portions of the auditory system. [72] For some ototoxic chemical exposures, particularly styrene, [73] the risk of hearing loss can be higher than being exposed to noise alone.
Nonsyndromic deafness constitutes 75% of all hearing loss cases, and an estimated 100 genes are thought to be linked to this condition. About 80% are linked to autosomal recessive inheritance, 15% to autosomal dominant inheritance, 1-3% through the X chromosome, and 0.5-1% are associated with mitochondrial inheritance .
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