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Erythema annulare centrifugum (EAC), is a descriptive term for a class of skin lesion [2] presenting redness in a ring form (anulare) that spreads from a center (centrifugum). It was first described by Darier in 1916.
Annular erythema of infancy (AEI) consists of self-limited eruptions of erythematous, annular to polycyclic patches and plaques. It is an idiopathic figurate erythema. [1] Over several days, a single lesion disappears without leaving behind any scale or hyperpigmentation. Mostly affecting the trunk, face, and extremities, this rash has no symptoms.
Erythema annulare centrifugum (deep gyrate erythema, erythema perstans, palpable migrating erythema, superficial gyrate erythema) Erythema gyratum repens (Gammel's disease) Erythema migrans (erythema chronicum migrans) Erythema multiforme; Erythema multiforme minor (herpes simplex-associated erythema multiforme) Erythema palmare; Generalized ...
Figurate erythema is a form of erythema (reddening of the skin) that presents in a ring or an arc shape. An example is erythema marginatum. [1] Classical types include: [2] Erythema annulare centrifugum; Erythema marginatum rheumaticum; Erythema chronicum migrans; Erythema gyratum repens
In a normal case, the swelling will decrease without treatment within 15–30 minutes, but, in extreme cases, itchy red welts may last anywhere from a few hours to days. In some cases, welts are accompanied with a painful burning sensation. [3] This calls for more urgent treatment as the condition can impact on the patient's quality of life.
Erythema multiforme (EM) is generally considered a separate condition. [6] Treatment typically takes place in hospital such as in a burn unit or intensive care unit. [3] [7] Efforts include stopping the cause, pain medication, and antihistamines. [3] [4] Antibiotics, intravenous immunoglobulins, and corticosteroids may also be used.
It is used to describe the rash in cutaneous larvae migrans, [3] erythema annulare centrifugum, [4] purpura annularis telangiectoides, ringworm, [5] balanitis circinata, [6] and some cases of bullous pemphigoid. [7]
Common causes of allergic contact dermatitis include: nickel allergy, 14K or 18K gold, Balsam of Peru (Myroxylon pereirae), and chromium. In the Americas they include the oily, urushiol-containing coating from plants of the genus Toxicodendron: poison ivy, poison oak, and poison sumac. Millions of cases occur each year in North America alone. [10]