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The optic radiation contains feedforward tracts that transmit visual information (from the retina of the eye) from the geniculate nucleus to the visual cortex, and also feedback tracts from the visual cortex to the neurons in the LGN that project to them. [2] The function of the feedback from the visual cortex back to the LGN is unknown.
This is most commonly the result of exposure to the sun at high altitude, and in areas where shorter wavelengths are readily reflected from bright surfaces, such as snow, water, and sand. UV generated by a welding arc can similarly cause damage to the cornea, known as "arc eye" or welding flash burn, a form of photokeratitis. [52]
Rate limits characterize the exposure based on effective energy (radiance or irradiance, depending on the type of radiation and the health effect of concern) per area per time, and dose limits characterize the exposure as a total acceptable dose. The latter is applied when the intensity of the radiation is great enough to produce a thermal injury.
Radiation is a moving form of energy, classified into ionizing and non-ionizing type. [4] Ionizing radiation is further categorized into electromagnetic radiation (without matter) and particulate radiation (with matter). [4] Electromagnetic radiation consists of photons, which can be thought of as energy packets, traveling in the form of a wave ...
The near-infrared (NIR) window (also known as optical window or therapeutic window) defines the range of wavelengths from 650 to 1350 nanometre (nm) where light has its maximum depth of penetration in tissue. [1] Within the NIR window, scattering is the most dominant light-tissue interaction, and therefore the propagating light becomes diffused ...
Ischemic optic neuropathies are classified based on the location of the damage and the cause of reduced blood flow if known. [3] Anterior ischemic optic neuropathy (AION) includes diseases that affect the optic nerve head and cause swelling of the optic disc. These diseases often cause sudden rapid visual loss in one eye.
Other factors can cause glaucoma, known as "secondary glaucoma", including prolonged use of steroids (steroid-induced glaucoma); conditions that severely restrict blood flow to the eye, such as severe diabetic retinopathy and central retinal vein occlusion (neovascular glaucoma); ocular trauma (angle-recession glaucoma); plateau iris; and ...
The blood–ocular barrier is a barrier created by endothelium of capillaries of the retina and iris, ciliary epithelium and retinal pigment epithelium. [1] It is a physical barrier between the local blood vessels and most parts of the eye itself, and stops many substances including drugs from traveling across it. [2]