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Vibrio alginolyticus is a Gram-negative marine bacterium. [1] [2] It is medically important since it causes otitis and wound infection. [1]It is also present in the bodies of animals such as pufferfish, where it is responsible for the production of the potent neurotoxin, tetrodotoxin.
Dermocystidium erschowii Garkavi, Denisov & Afanas'ev 1980— a skin parasite of carp; Dermocystidium fennicum Pekkarinen et al. 2003— a skin parasite of perch [9] Dermocystidium gasterostei Elkan 1962 [7] — a parasite of sticklebacks [10] Dermocystidium granulosum Sterba & Naumann 1970 [7] Dermocystidium guyenotii Thélin 1955 [7]
Non-specific defences include skin and scales, as well as the mucus layer secreted by the epidermis that traps microorganisms and inhibits their growth. If pathogens breach these defences, fish can develop inflammatory responses that increase the flow of blood to infected areas and deliver white blood cells that attempt to destroy the pathogens.
Edwardsiella tarda is a member of the family Hafniaceae. [1] [2] The bacterium is a facultatively anaerobic, small, motile, gram negative, straight rod with flagella.[1] [2] Infection causes Edwardsiella septicemia (also known as ES, edwardsiellosis, emphysematous putrefactive disease of catfish, fish gangrene, and red disease) in channel catfish, eels, and flounder.
Some fish show no external symptoms, but others show signs of infection that include bulging eyes, bloated abdomens, bruised-looking reddish tints to the eyes, skin, gills and fins. Some infected fish have open sores that may look like the lesions from other diseases or from lamprey attacks.
The lesions observed on fish presumed killed by Pfiesteria have been attributed to water molds by some researchers. However, it has also been established that Pfiesteria shumwayae kills fish by feeding on their skin through myzocytosis. [11] In early 2007, a highly unstable toxin produced by the toxic form of Pfiesteria piscicida was identified ...
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Alternatively, a fish can become infected by eating an infected oligochaete. Infected tubificids can release triactinomyxons for at least a year. The triactinomyxon spores are carried by the water currents, where they can infect a salmonid through the skin. Penetration of the fish by these spores takes only a few seconds.