Search results
Results from the WOW.Com Content Network
The superior thalamic vein (Latin: vena superioris thalami), initially called by Benno Shlesinger in 1976 the principal thalamic vein (vena principalis thalami) or centro-medial thalamic vein (vena centro-medialis thalami), also called by Russian surgeon Pirogoff internal thalamic vein (vena interioris thalami) is the most prominent vein of the thalamus.
3D model of cerebral veins. In human anatomy, the cerebral veins are blood vessels in the cerebral circulation which drain blood from the cerebrum of the human brain.They are divisible into external (superficial cerebral veins) and internal (internal cerebral veins) groups according to the outer or inner parts of the hemispheres they drain into.
The venous drainage of the cerebrum can be separated into two subdivisions: superficial and deep. The superficial system. The superficial system is composed of dural venous sinuses, sinuses (channels) within the dura mater. The dural sinuses are therefore located on the surface of the cerebrum.
The superior thalamostriate vein or terminal vein commences in the groove between the corpus striatum and thalamus, receives numerous veins from both of these parts, and unites behind the crus of the fornix with the superior choroid vein to form each of the internal cerebral veins.
A developmental venous anomaly (DVA, formerly known as venous angioma) is a congenital variant of the cerebral venous drainage. On imaging it is seen as a number of small deep parenchymal veins converging toward a larger collecting vein.
The pathophysiology of cranial venous outflow obstruction involves the disruption of normal venous drainage from the brain. Cerebral veins play a crucial role in draining brain interstitial fluid (ISF), and their significance has been linked in various neurological conditions. [1] It can be caused by extrinsic or intrinsic anomalies. [7]
The most frequently observed problems related to a cerebral arteriovenous malformation (AVM) are headaches and seizures, cranial nerve afflictions including pinched nerve and palsy, [2] [3] backaches, neckaches, and nausea from coagulated blood that has made its way down to be dissolved in the cerebrospinal fluid.
A side effect of this is that it could lower pressure of blood to the head, resulting in a reduced and possibly inadequate blood supply to the brain. Venous drainage may also be impeded by external factors such as hard collars to immobilize the neck in trauma patients, and this may also increase the ICP.