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  2. Hypokalemia - Wikipedia

    en.wikipedia.org/wiki/Hypokalemia

    Hypokalemia which is recurrent or resistant to treatment may be amenable to a potassium-sparing diuretic, such as amiloride, triamterene, spironolactone, or eplerenone. Concomitant hypomagnesemia will inhibit potassium replacement, as magnesium is a cofactor for potassium uptake.

  3. Electrolyte imbalance - Wikipedia

    en.wikipedia.org/wiki/Electrolyte_imbalance

    [3] [27] Once the patient is stable, it is important to identify the underlying cause of hypernatremia as that may affect the treatment plan. [3] [27] The final step in treatment is to calculate the patients free water deficit, and to replace it at a steady rate using a combination of oral or IV fluids.

  4. Hypokalemic periodic paralysis - Wikipedia

    en.wikipedia.org/wiki/Hypokalemic_periodic_paralysis

    Hypokalemic periodic paralysis is a condition that causes episodes of extreme muscle weakness typically beginning in childhood or adolescence. Most often, these episodes involve a temporary inability to move muscles in the arms and legs. Attacks cause severe weakness or paralysis that usually lasts from hours to days.

  5. Distal renal tubular acidosis - Wikipedia

    en.wikipedia.org/wiki/Distal_renal_tubular_acidosis

    dRTA commonly leads to sodium loss and volume contraction, which causes a compensatory increase in blood levels of aldosterone. [4] Aldosterone causes increased resorption of sodium and loss of potassium in the collecting duct of the kidney, so these increased aldosterone levels cause the hypokalemia which is a common symptom of dRTA. [4]

  6. Gitelman syndrome - Wikipedia

    en.wikipedia.org/wiki/Gitelman_syndrome

    Gitelman syndrome; Other names: Primary renal tubular hypokalemic hypomagnesemia with hypocalciuria: A model of transport mechanisms in the distal convoluted tubule.Sodium chloride (NaCl) enters the cell via the apical thiazide-sensitive NCC and leaves the cell through the basolateral Cl − channel (ClC-Kb), and the Na + /K +-ATPase.

  7. Diabetic ketoacidosis - Wikipedia

    en.wikipedia.org/wiki/Diabetic_ketoacidosis

    Hypokalemia (low blood potassium concentration) often follows treatment. This increases the risk of dangerous irregularities in the heart rate . Therefore, continuous observation of the heart rate is recommended, [ 6 ] [ 39 ] as well as repeated measurement of the potassium levels and addition of potassium to the intravenous fluids once levels ...

  8. Parenteral nutrition - Wikipedia

    en.wikipedia.org/wiki/Parenteral_nutrition

    Metabolic complications include the refeeding syndrome characterised by hypokalemia, hypophosphatemia and hypomagnesemia. Hyperglycemia is common at the start of therapy, but can be treated with insulin added to the TPN solution. Hypoglycaemia is likely to occur with abrupt cessation of TPN.

  9. Nephrogenic diabetes insipidus - Wikipedia

    en.wikipedia.org/wiki/Nephrogenic_diabetes_insipidus

    Nephrogenic diabetes insipidus is most common in its acquired forms, meaning that the defect was not present at birth. These acquired forms have numerous potential causes. The most obvious cause is a kidney or systemic disorder, including amyloidosis, [2] polycystic kidney disease, [3] electrolyte imbalance, [4] [5] or some other kidney defect. [2]