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Gait abnormality is a deviation from normal walking ().Watching a patient walk is an important part of the neurological examination. Normal gait requires that many systems, including strength, sensation and coordination, function in an integrated fashion.
Gait freezing, also called motor blocks, is a manifestation of akinesia. [1] Gait freezing is characterized by a sudden inability to move the lower extremities which usually lasts less than 10 seconds. [1] It may worsen in tight, cluttered spaces, when attempting to initiate gait or turning around, or when approaching a destination. [1]
Parkinsonian gait (or festinating gait, from Latin festinare [to hurry]) is the type of gait exhibited by patients with Parkinson's disease (PD). [2] It is often described by people with Parkinson's as feeling like being stuck in place, when initiating a step or turning, and can increase the risk of falling. [ 3 ]
These paravertebral muscles have a great influence over the walking stance and gait of a patient, so fatty infiltration and degradation of these muscle lead to the characteristics that easily define BSS, such as the anterior flexion of the back combined with an ability to keep upright with any kind of support (e.g., holding onto a table).
Gait is the way in which you walk. While everyone’s gait differs, you may develop an abnormal gait due to illness, injury, genetics, or issues with your legs or feet.
Myopathic gait (or waddling gait) is a form of gait abnormality. The "waddling" is due to the weakness of the proximal muscles of the pelvic girdle. [1]
Steppage gait (high stepping, neuropathic gait) is a form of gait abnormality characterised by foot drop or ankle equinus due to loss of dorsiflexion. [1] The foot hangs with the toes pointing down, causing the toes to scrape the ground while walking, requiring someone to lift the leg higher than normal when walking.
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