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Seleucus I Nicator, the first Macedonian king of Syria, was said to have received from Apollo an anchor-shaped birthmark on his thigh, which was also borne by his descendants. A short Greek manual of mole interpretation survives from antiquity, appended to the end of a much longer work on divination by twitches ascribed to the legendary Greek ...
Mongolian spot is a congenital developmental condition—that is, one existing from birth—exclusively involving the skin.The blue colour is caused by melanocytes, melanin-containing cells, that are usually located in the surface of the skin (the epidermis), but are in the deeper region (the dermis) in the location of the spot. [6]
Several birthmark types are part of the group of skin lesions known as nevi or naevi, which is Latin for "birthmarks". Birthmarks occur as a result of a localized imbalance in factors controlling the development and migration of skin cells. In addition, it is known that vascular birthmarks are not hereditary. [2] [3]
It should not be confused with Mongolian spot, which is a birthmark caused by entrapment of melanocytes in the dermis but is located in the lumbosacral region. Women are nearly five times more likely to be affected than men, and it is rare among Caucasian people. [6] Nevus of Ota may not be congenital, and may appear during puberty.
Congenital nevus. Note the variable coloration and slightly irregular border. The congenital melanocytic nevus appears as a circumscribed, light brown to black patch or plaque, potentially very heterogeneous in consistency, covering any size surface area and any part of the body.
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A beauty mark or beauty spot is a euphemism for a type of dark facial mark so named because such birthmarks are sometimes considered an attractive feature. [1] Medically, such "beauty marks" are generally melanocytic nevus, more specifically the compound variant. Moles of this type may also be located elsewhere on the body, and may also be ...
Autosomal dominant A 50/50 chance of inheritance. Sickle-cell disease is inherited in the autosomal recessive pattern. When both parents have sickle-cell trait (carrier), a child has a 25% chance of sickle-cell disease (red icon), 25% do not carry any sickle-cell alleles (blue icon), and 50% have the heterozygous (carrier) condition. [1]