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The five main types of psoriasis are plaque, guttate, inverse, pustular, and erythrodermic. [5] Plaque psoriasis, also known as psoriasis vulgaris, makes up about 90% of cases. [4] It typically presents as red patches with white scales on top. [4] Areas of the body most commonly affected are the back of the forearms, shins, navel area, and ...
These rashes are often very red, macerated, and erosive-looking and are accompanied by red bumps or papules. Other symptoms to note : Candida rashes often have a “sour, yeasty odor,” Dr. Lal ...
Psoriasis is a common, chronic, and recurrent inflammatory disease of the skin characterized by circumscribed, erythematous, dry, scaling plaques. [ 97 ] [ 98 ] [ 99 ] Psoriasis vulgaris
Discoid lupus erythematosus is the most common type of chronic cutaneous lupus (CCLE), an autoimmune skin condition on the lupus erythematosus spectrum of illnesses. [1] [2] It presents with red, painful, inflamed and coin-shaped patches of skin with a scaly and crusty appearance, most often on the scalp, cheeks, and ears.
Wheal: A wheal is a rounded or flat-topped, pale red papule or plaque that is characteristically evanescent, disappearing within 24 to 48 hours. The temporary raised skin on the site of a properly delivered intradermal (ID) injection is also called a welt, with the ID injection process itself frequently referred to as simply "raising a wheal ...
Erythema (Ancient Greek: ἐρύθημα, from Greek erythros 'red') is redness of the skin or mucous membranes, caused by hyperemia (increased blood flow) in superficial capillaries. [1] It occurs with any skin injury, infection, or inflammation. Examples of erythema not associated with pathology include nervous blushes. [2]
Parakeratosis is seen in the plaques of psoriasis and in dandruff. Granular parakeratosis (originally termed axillary granular parakeratosis) is an idiopathic, benign, nondisabling cutaneous disease that manifests with intertriginous erythematous, brown or red, scaly or keratotic papules and plaques. It presents in all age groups and has no ...
There is little evidence to suggest that the pathophysiologic mechanisms underlying inverse and common psoriasis vary from one another. The amount of CD161+ cells in the inverse psoriasis plaques, however, might be diminishing. [3] This is thought to be caused by the ongoing microbial colonization of the inverse psoriasis-affected regions. [5]