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Moraxella lacunata was first described independently by Victor Morax (1896) and Theodor Axenfeld (1897), hence the alternate name "Morax-Axenfeld diplobacilli" and the name of eye infection in humans is sometimes called Morax-Axenfeld conjunctivitis.
Conjunctivitis due to common pus-producing bacteria causes marked grittiness or irritation and a stringy, opaque, greyish or yellowish discharge that may cause the lids to stick together, especially after sleep. Severe crusting of the infected eye and the surrounding skin may also occur.
In the anatomy of the eye, the conjunctiva (pl.: conjunctivae) is a thin mucous membrane that lines the inside of the eyelids and covers the sclera (the white of the eye). [1] It is composed of non-keratinized, stratified squamous epithelium with goblet cells, stratified columnar epithelium and stratified cuboidal epithelium (depending on the ...
"Conjunctivitis basically means inflammation of the conjunctiva, which is the clear part that covers the white part of the eyes," says Dr. Sumitra Khandelwal, an associate professor of ...
Types include sympathetic ophthalmia (inflammation of both eyes following trauma to one eye), gonococcal ophthalmia, trachoma or "Egyptian" ophthalmia, ophthalmia neonatorum (a conjunctivitis [3] of the newborn due to either of the two previous pathogens), photophthalmia and actinic conjunctivitis (inflammation resulting from prolonged exposure to ultraviolet rays), and others.
Actinic conjunctivitis is an inflammation of the eye contracted from prolonged exposure to actinic (ultraviolet) rays. Symptoms are redness and swelling of the eyes. Most often the condition is caused by prolonged exposure to Klieg lights, therapeutic lamps or acetylene torches. Other names for the condition include Klieg conjunctivitis ...
Trachoma is an infectious disease caused by bacterium Chlamydia trachomatis. [2] The infection causes a roughening of the inner surface of the eyelids. [2] This roughening can lead to pain in the eyes, breakdown of the outer surface or cornea of the eyes, and eventual blindness. [2]
The epidemic nature of this bacteria has been seen in the high frequency of “control” subjects from the affected areas of Brazil that have or had recently had conjunctivitis. These control subjects did not develop Brazilian Purpuric Fever, and therefore were probably not carrying the more dangerous BPF clone of H. influenzae biogroup aegyptius.