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Serum sickness in humans is a reaction to proteins in antiserum derived from a non-human animal source, occurring 5–10 days after exposure. Symptoms often include a rash , joint pain , fever , and lymphadenopathy .
On physical exam, kwashiorkor is also distinguished from marasmus by the presence of edema. When children present with both kwashiorkor and marasmus, the condition is referred to as "marasmic-kwashiorkor". [11] [3] In general, kwashiorkor is marked by more profound serum depletions of antioxidant molecules and minerals, relative to marasmus. [3]
Map of the historical and current Iditarod trails; the route taken during the 1925 serum run is shown in green.. The 1925 serum run to Nome, also known as the Great Race of Mercy and The Serum Run, was a transport of diphtheria antitoxin by dog sled relay across the US territory of Alaska by 20 mushers and about 150 sled dogs across 674 miles (1,085 km) in 5 + 1 ⁄ 2 days, saving the small ...
As with IgA nephropathy, serum levels of IgA are high in HSP and there are identical findings on renal biopsy; however, IgA nephropathy has a predilection for young adults while HSP is more predominant among children. Further, IgA nephropathy typically only affects the kidneys while HSP is a systemic disease.
In 1906, Clemens Pirquet and Béla Schick described serum sickness in children receiving large quantities of horse-derived antitoxin. [52] Between 1910 and 1911, Béla Schick developed the Schick test to detect pre-existing immunity to diphtheria in an exposed person. Only those who had not been exposed to diphtheria were vaccinated.
Serum sickness–like reaction; Setleis syndrome; Severe acute respiratory syndrome; Shaken baby syndrome; Shapiro syndrome; Sheehan's syndrome; Shell nail syndrome; Shone's syndrome; Short anagen syndrome; Short bowel syndrome; Short man syndrome; Short QT syndrome; Short rib – polydactyly syndrome; SHORT syndrome; Shwachman–Diamond ...
Since the syndrome is due to the accumulation of chloramphenicol, the signs and symptoms are dose related. [10] According to Kasten's review published in the Mayo Clinic Proceedings, a serum concentration of more than 50 μg/mL is a warning sign, [10] while Hammett-Stabler and John states that the common therapeutics peak level is 10-20 μg/mL and is expected to achieve after 0.5-1.5 hours of ...
Serum sickness-like reaction; Steroid acne; Steroid folliculitis; Stevens–Johnson syndrome; Sulfonamide hypersensitivity syndrome; Texier's disease; Toxic epidermal necrolysis (Lyell's syndrome) Urticarial erythema multiforme; Vitamin K reaction; Warfarin necrosis