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Contrast-induced nephropathy (CIN) is a purported form of kidney damage in which there has been recent exposure to medical imaging contrast material without another clear cause for the acute kidney injury. Despite extensive speculation, the actual occurrence of contrast-induced nephropathy has not been demonstrated in the literature. [1]
In 2006, the link between NSF and gadolinium-based MRI contrast agents was made. [ 4 ] [ 23 ] [ 24 ] As a result, restrictions on use of GBCAs in patients with an estimated glomerular filtration rate (a measure of kidney function ) under 60 and especially under 30 mL/min/1.73 m 2 have been recommended and NSF is now considered rare.
Acute kidney injury was one of the most expensive conditions seen in U.S. hospitals in 2011, with an aggregated cost of nearly $4.7 billion for approximately 498,000 hospital stays. [48] This was a 346% increase in hospitalizations from 1997, when there were 98,000 acute kidney injury stays. [49]
Kidney infarction: CT scan of the abdomen showing partial infarct of the left kidney. Specialty: Nephrology: Symptoms: Abdominal pain, nausea, vomiting, and fever. [1] Complications: Acute kidney injury and chronic kidney disease. [1] Causes: Cardioembolic disease, renal artery injury, and hypercoagulable state. [1] Diagnostic method
It may cause acute kidney injury. It is now more commonly used to describe diffuse, fine, renal parenchymal calcification in radiology. [2] It is caused by multiple different conditions and is determined by progressive kidney dysfunction. These outlines eventually come together to form a dense mass. [3]
They learned that people who had acute kidney injuries had a 49% higher chance of dementia than people who did not have an acute kidney injury. The study appears in the journal Neurology .
Renal cortical necrosis (RCN) is a rare cause of acute kidney failure.The condition is "usually caused by significantly diminished arterial perfusion of the kidneys due to spasms of the feeding arteries, microvascular injury, or disseminated intravascular coagulation" and is the pathological progression of acute tubular necrosis. [1]
Acute tubular necrosis (ATN) is a medical condition involving the death of tubular epithelial cells that form the renal tubules of the kidneys. Because necrosis is often not present, the term acute tubular injury ( ATI ) is preferred by pathologists over the older name acute tubular necrosis (ATN). [ 1 ]