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The nucleus accumbens (NAc or NAcc; also known as the accumbens nucleus, or formerly as the nucleus accumbens septi, Latin for 'nucleus adjacent to the septum') is a region in the basal forebrain rostral to the preoptic area of the hypothalamus. [1]
The nucleus accumbens (NAcc) is located in the basal forebrain and is a major structure of the ventral striatum, an area of the brain associated with the limbic system, and regulates limbic functions of motivation, affect, and reward.
The nucleus accumbens functions as an important modulation center between the limbic and the motor systems because of its input from the limbic system and its output to motor nuclei of the basal ganglia. The nucleus accumbens is involved in various cognitive, emotional, and psychomotor functions.
A target of increasing interest is the nucleus accumbens (NAc), which has long been studied as a key brain region mediating a variety of behaviors, including reward and satisfaction.
The nucleus accumbens (NAc) is a subcortical brain structure known primarily for its roles in pleasure, reward, and addiction. Despite less focus on the NAc in pain research, it also plays a large role in the mediation of pain and is effective as a source of analgesia.
The nucleus accumbens (NAc) is a key node of the brain’s circuitry that is responsible for translating motivation into action. It has been implicated in playing critical roles in virtually all forms of adaptive and pathological motivated behaviors.
The nucleus accumbens is a stucture in the rostrobasal forebrain that for at least the past twenty years has been regarded by the neuroscientific community as a highly differentiated, striatal (i.e. input nucleus) part of the ventral striatopallidal system [65–67].