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William Tallant Greenough (October 11, 1944 – December 18, 2013) was a professor of psychology at the University of Illinois at Urbana–Champaign.Greenough was a pioneer in studies of neural development and brain plasticity.
David Eagleman (born April 25, 1971) is an American neuroscientist, author, and science communicator.He teaches neuroscience at Stanford University [1] and is CEO and co-founder of Neosensory, a company that develops devices for sensory substitution. [2]
Paul Bach-y-Rita (April 4, 1934 – November 20, 2006) was an American neuroscientist whose most notable work was in the field of neuroplasticity.Bach-y-Rita was one of the first to seriously study the idea of neuroplasticity (although it was first proposed in the late 19th century), and to introduce sensory substitution as a tool to treat patients with neurological disorders.
How the brain changes. Brain plasticity science is the study of a physical process. Gray matter can actually shrink or thicken; neural connections can be forged and refined or weakened and severed.
Neuroplasticity, also known as neural plasticity or just plasticity, is the ability of neural networks in the brain to change through growth and reorganization. Neuroplasticity refers to the brain's ability to reorganize and rewire its neural connections, enabling it to adapt and function in ways that differ from its prior state.
Developmental plasticity is a general term referring to changes in neural connections during development as a result of environmental interactions as well as neural changes induced by learning. [1] Much like neuroplasticity , or brain plasticity, developmental plasticity is specific to the change in neurons and synaptic connections as a ...
The Adult Plasticity period is when the brain refines its neural processes as it masters a variety of tasks. [ 24 ] [ 25 ] Understanding how the brain can re-wire itself has allowed Merzenich, Tallal, and other colleagues to develop strategies intended to remediate individuals with any speech, language, and reading deficits.
Two molecular mechanisms for synaptic plasticity involve the NMDA and AMPA glutamate receptors. Opening of NMDA channels (which relates to the level of cellular depolarization) leads to a rise in post-synaptic Ca 2+ concentration and this has been linked to long-term potentiation, LTP (as well as to protein kinase activation); strong depolarization of the post-synaptic cell completely ...