Search results
Results from the WOW.Com Content Network
Vitamin D compounds, specifically cholecalciferol (D3) and ergocalciferol (D2), are used in rodenticides due to their ability to induce hypercalcemia, a condition characterized by elevated calcium levels in the blood. This overdose leads to organ failure and is pharmacologically similar to vitamin D's toxic effects in humans.
A surplus of vitamin D, though, can be dangerous. ... considers a reading above 125 nmol/L high enough to cause health problems. ... can include stupor, coma, kidney stones, stomach ulcers, nausea ...
She urges caution because vitamin D can build up to toxic levels if taken in excess for too long. It can cause calcium to build up too high, contributing to kidney stones and even bone damage ...
This condition can be caused by vitamin D, calcium or phosphorus deficiency. [69] Vitamin D deficiency remains the main cause of rickets among young infants in most countries because breast milk is low in vitamin D, and darker skin, social customs, and climatic conditions can contribute to inadequate sun exposure.
"abdominal pain, diarrhea, potentially carcinogenic, with others can potentiate cardiac glycosides and antiarrhythmic agents" [3] Areca nut: betel nut Areca catechu "deterioration of psychosis in patients with preexisting psychiatric disorders"; [5] known carcinogen contributing to cancer of the mouth, pharynx, esophagus and stomach when chewed ...
Vitamin D is the all encompassing name for a family of similar chemical components needed in the human body, says Courtney Pelitera, M.S., R.D., C.N.S.C., registered dietitian and owner of Devour ...
Hypervitaminosis is a condition of abnormally high storage levels of vitamins, which can lead to various symptoms as over excitement, irritability, or even toxicity. Specific medical names of the different conditions are derived from the given vitamin involved: an excess of vitamin A, for example, is called hypervitaminosis A.
The cause of IBS is not known but multiple factors have been proposed to lead to the condition. [2] Theories include combinations of "gut–brain axis" problems, alterations in gut motility, visceral hypersensitivity, infections including small intestinal bacterial overgrowth, neurotransmitters, genetic factors, and food sensitivity. [2]