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A heart attack occurs suddenly when an atherosclerotic plaque in one of the arteries to your heart ruptures. It can cause symptoms such as: Chest pain, pressure, or tightness
Coronary artery disease (CAD) or ischemic heart disease are the terms used to describe narrowing of the coronary arteries. [8] As the disease progresses, plaque buildup can partially block blood flow to the heart muscle. Without enough blood supply , the heart is unable to work properly, especially under increased stress.
The relatively narrow coronary arteries are commonly affected by atherosclerosis and can become blocked, causing angina or a heart attack. The coronary arteries are classified as "terminal circulation", since they represent the only source of blood supply to the myocardium; there is very little redundant blood supply, that is why blockage of ...
The animation shows plaque buildup or a coronary artery spasm can lead to a heart attack and how blocked blood flow in a coronary artery can lead to a heart attack. The most common cause of a myocardial infarction is the rupture of an atherosclerotic plaque on an artery supplying heart muscle.
"Dental plaque is a sticky film of bacteria, while arterial plaque is a complex buildup of cholesterol, fat, calcium and cellular debris on artery walls," explains Dr. Rigved Tadwalkar, MD, a ...
The most common type is coronary artery disease, in which plaque builds up in the arteries and blocks blood flow to the heart, potentially leading to heart attacks or heart failure.
Arteriosclerosis, literally meaning "hardening of the arteries", is an umbrella term for a vascular disorder characterized by abnormal thickening, hardening, and loss of elasticity [3] of the walls of arteries; [4] this process gradually restricts the blood flow to one's organs and tissues and can lead to severe health risks brought on by atherosclerosis, which is a specific form of ...
The fibro-lipid (fibro-fatty) plaque is characterized by an accumulation of lipid-laden cells underneath the intima of the arteries, typically without narrowing the lumen due to compensatory expansion of the bounding muscular layer of the artery wall. Beneath the endothelium, there is a "fibrous cap" covering the atheromatous "core" of the plaque.
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