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  2. Equine metabolic syndrome - Wikipedia

    en.wikipedia.org/wiki/Equine_metabolic_syndrome

    Diagnosis is based on history, clinical signs, and diagnostic tests. Glucose levels alone are insufficient to diagnose EMS. Many EMS horses will effectively compensate their insulin response even with insulin resistance, maintaining a blood glucose within normal limits, although usually at the high end of normal.

  3. Hypokalemia - Wikipedia

    en.wikipedia.org/wiki/Hypokalemia

    Hypokalemia is a low level of potassium (K +) in the blood serum. [1] Mild low potassium does not typically cause symptoms. [3] Symptoms may include feeling tired, leg cramps, weakness, and constipation. [1] Low potassium also increases the risk of an abnormal heart rhythm, which is often too slow and can cause cardiac arrest. [1] [3]

  4. Hypokalemic periodic paralysis - Wikipedia

    en.wikipedia.org/wiki/Hypokalemic_periodic_paralysis

    Hypokalemic periodic paralysis (hypoKPP), also known as familial hypokalemic periodic paralysis (FHPP), [1] is a rare, autosomal dominant channelopathy characterized by muscle weakness or paralysis when there is a fall in potassium levels in the blood (hypokalemia).

  5. Pituitary pars intermedia dysfunction - Wikipedia

    en.wikipedia.org/wiki/Pituitary_pars_intermedia...

    In PPID-affected horses, dopamine is not produced from these neurons, leading to dopamine levels about 10% of the level normally found in the pars intermedia. [2] This is thought to be due to neurodegeneration of the neurons, secondary to free radical formation and oxidative stress. [3]

  6. Electrolyte imbalance - Wikipedia

    en.wikipedia.org/wiki/Electrolyte_imbalance

    Low potassium is caused by increased excretion of potassium, decreased consumption of potassium rich foods, movement of potassium into the cells, or certain endocrine diseases. [3] Excretion is the most common cause of hypokalemia and can be caused by diuretic use, metabolic acidosis , diabetic ketoacidosis , hyperaldosteronism , and renal ...

  7. Gitelman syndrome - Wikipedia

    en.wikipedia.org/wiki/Gitelman_syndrome

    Gitelman syndrome; Other names: Primary renal tubular hypokalemic hypomagnesemia with hypocalciuria: A model of transport mechanisms in the distal convoluted tubule.Sodium chloride (NaCl) enters the cell via the apical thiazide-sensitive NCC and leaves the cell through the basolateral Cl − channel (ClC-Kb), and the Na + /K +-ATPase.

  8. Proximal renal tubular acidosis - Wikipedia

    en.wikipedia.org/wiki/Proximal_renal_tubular...

    Patients with type 2 RTA are also typically hypokalemic due to a combination of secondary hyperaldosteronism, and potassium urinary losses - though serum potassium levels may be falsely elevated because of acidosis. Administration of bicarbonate prior to potassium supplementation might lead to worsened hypokalemia, as potassium shifts ...

  9. Limbic-predominant age-related TDP-43 encephalopathy

    en.wikipedia.org/wiki/Limbic-predominant_age...

    LATE is a term that describes a prevalent medical condition with impaired memory and thinking in advanced age, often culminating in the dementia clinical syndrome. [1] In other words, the symptoms of LATE are similar to those of Alzheimer's disease. The acronym LATE stands for Limbic-predominant Age-related TDP-43 Encephalopathy.