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The signs and symptoms of hydronephrosis depend upon whether the obstruction is acute or chronic, partial or complete, unilateral or bilateral.Hydronephrosis that occurs acutely with sudden onset (as caused by a kidney stone) can cause intense pain in the flank area (between the hips and ribs) known as a renal colic.
A hydronephrotic kidney may present as a palpable abdominal mass in the newborn, and may suggest an ectopic ureter or ureterocele. In older children, ureteral duplication may present as: [citation needed] Urinary tract infection – most commonly due to vesicoureteral reflux (flow of urine from the bladder into the ureter, rather than vice versa).
Generally, diseases outlined within the ICD-10 codes N00-N19, N20.0 within Chapter XIV: Diseases of the genitourinary system should be included in this category. Wikimedia Commons has media related to Kidney diseases .
Characteristics of Some Inherited Tubulopathies [4]; Disorder [OMIM Number] Protein Defect Chromosome Localization Inheritance Clinical Features/Notes Biochemical Features
The kidney is responsible for excreting nitrogenous substances in the urine, so an elevated BUN usually indicates that the kidney is not functioning appropriately. [ 32 ] Urinalysis - After the patient provides a urine specimen , it is sent to the lab for analysis using a variety of methods including urine dipstick testing and microscopic ...
The symptoms of chronic syndrome of inappropriate antidiuresis are more vague, and may include cognitive impairment, gait abnormalities, or osteoporosis. [2] The main treatment of inappropriate antidiuresis is to identify and treat the underlying cause, if possible.
Acute tubular necrosis (ATN) is a medical condition involving the death of tubular epithelial cells that form the renal tubules of the kidneys.Because necrosis is often not present, the term acute tubular injury (ATI) is preferred by pathologists over the older name acute tubular necrosis (ATN). [1]
Membranoproliferative glomerulonephritis (MPGN) is a type of glomerulonephritis caused by deposits in the kidney glomerular mesangium and basement membrane thickening, [2] activating the complement system and damaging the glomeruli. MPGN accounts for approximately 4% of primary renal causes of nephrotic syndrome in children and 7% in adults. [3]