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In 2011, Porrello et al. demonstrated that neonatal mice are able to regenerate heart muscle after resection. [11] Since 2011, many other research groups have explored cardiomyocyte regeneration. The cardiomyocytes of neonatal rats [ 12 ] and piglets [ 13 ] are also able to undergo proliferation in response to injury during the first week of life.
The heart has the potential to repair itself when damaged using progenitor and stem cells. [10] Clinical trials have shown that heart muscle has not previously been able to regenerate itself. New noninvasive drugs, which may make this possible in humans, are required to induce the cardiac myocytes to proliferate.
Inhibition of p38 MAP kinase was found to induce mitosis in adult mammalian cardiomyocytes, [49] while treatment with FGF1 and p38 MAP kinase inhibitors was found to regenerate the heart, reduce scarring, and improve cardiac function in rats with cardiac injury. [50] One of the most promising sources of heart regeneration is the use of stem cells.
A syndrome is a set of medical signs and symptoms that are correlated with each other. A syndrome can affect one or more of body systems. Different syndromes affect different groups of organs. This is a list of syndromes that may affect the heart. Syndromes affecting primarily the heart are written in bold letters. [1] [2]
The existing epithelial cells can replicate, and, using the basement membrane as a guide, eventually bring the kidney back to normal. After regeneration is complete, the damage is undetectable, even microscopically. [citation needed] Healing must happen by repair in the case of injury to cells that are unable to regenerate (e.g. neurons).
Dr. Kevin Watt, team leader of the Heart Regeneration and Disease Laboratory at the Murdoch Children’s Research Institute (MCRI) in Melbourne, Australia, understands this concept deeply.
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