Search results
Results from the WOW.Com Content Network
Hematidrosis, also called hematohidrosis, haematidrosis, hemidrosis and blood sweat, is a very rare condition in which a human sweats blood. [1] The term is from Ancient Greek haîma / haímatos ( αἷμα / αἵματος ), meaning blood, and hīdrṓs ( ἱδρώς ), meaning sweat.
Hemosiderosis is a form of iron overload disorder resulting in the accumulation of hemosiderin.. Types include: Transfusion hemosiderosis [1]; Idiopathic pulmonary hemosiderosis
In terms of haemophilia C medication tranexamic acid is often used for both treatment after an incident of bleeding and as a preventive measure to avoid excessive bleeding during oral surgery. [ 3 ] Treatment is usually not necessary, except in relation to operations , leading to many of those having the condition not being aware of it.
Typical treatment for chronic iron overload requires subcutaneous injection over a period of 8–12 hours daily. [ citation needed ] Two newer iron-chelating drugs that are licensed for use in patients receiving regular blood transfusions to treat thalassaemia (and, thus, who develop iron overload as a result) are deferasirox and deferiprone .
The symptoms of DRESS syndrome usually begin 2 to 6 weeks but uncommonly up to 8–16 weeks after exposure to an offending drug. Symptoms generally include fever, an often itchy rash which may be morbilliform or consist mainly of macules or plaques, facial edema (i.e. swelling, which is a hallmark of the disease), enlarged and sometimes painful lymph nodes, and other symptoms due to ...
[19] [20] Clinically, the diagnosis of any particular skin condition is made by gathering pertinent information regarding the presenting skin lesion(s), including the location (such as arms, head, legs), symptoms (pruritus, pain), duration (acute or chronic), arrangement (solitary, generalized, annular, linear), morphology (macules, papules ...
Liddle's syndrome, also called Liddle syndrome, [1] is a genetic disorder inherited in an autosomal dominant manner that is characterized by early, and frequently severe, high blood pressure associated with low plasma renin activity, metabolic alkalosis, low blood potassium, and normal to low levels of aldosterone. [1]
The mechanism of action of nitisinone involves inhibition of 4-Hydroxyphenylpyruvate dioxygenase (HPPD). [5] [6] This is a treatment for patients with Tyrosinemia type 1 as it prevents the formation of 4-Maleylacetoacetic acid and fumarylacetoacetic acid, which have the potential to be converted to succinyl acetone, a toxin that damages the liver and kidneys. [4]