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Postpolypectomy coagulation syndrome (Postpolypectomy syndrome or PPCS) is a condition that occurs following colonoscopy with electrocautery polypectomy, which results in a burn injury to the wall of the gastrointestinal tract. The condition results in abdominal pain, fever, elevated white blood cell count and elevated serum C-reactive protein.
Symptoms of intestinal ischemia vary and can be acute (especially if embolic), [11] subacute, or chronic. [12] Case series report prevalence of clinical findings and provide the best available, yet biased, estimate of the sensitivity of clinical findings. [13] [14] In a series of 58 patients with intestinal ischemia due to mixed causes: [14]
Functional gastrointestinal disorders (FGID), also known as disorders of gut–brain interaction, include a number of separate idiopathic disorders which affect different parts of the gastrointestinal tract and involve visceral hypersensitivity and motility disturbances.
Primary vascular causes of bowel infarction, also known as mesenteric ischemia, are due to blockages in the arteries or veins that supply the bowel.Types of mesenteric ischemia are generally separated into acute and chronic processes, because this helps determine treatment and prognosis.
The lifelong risk of CRC is between 25 and 40%. SPS is the most common polyposis syndrome affecting the colon, but is under recognized due to a lack of systemic long term monitoring. [5] Diagnosis requires colonoscopy, and is defined by the presence of either of two criteria: five or more serrated lesions/polyps proximal to the rectum (all ≥ ...
Chronic prostatitis/chronic pelvic pain syndrome: CPDD Calcium pyrophosphate deposition disease: CPM Central pontine myelinolysis: CPPS Chronic pelvic pain syndrome (see UCPPS) CRE Carbapenem-resistant Enterobacteriaceae: CRF Chronic renal failure: CRKP Carbapenem-resistant Klebsiella pneumoniae: CRPS Complex regional pain syndrome: CSA Central ...
Gardner's syndrome (also known as Gardner syndrome, familial polyposis of the colon, [1] or familial colorectal polyposis [2]) is a subtype of familial adenomatous polyposis (FAP). Gardner syndrome is an autosomal dominant form of polyposis characterized by the presence of multiple polyps in the colon together with tumors outside the colon. [ 3 ]
This means the nerve signals are interrupted and are slower. This causes muscle contractions to be irregular and fewer, resulting in an increased colon transit time. [12] The feces stay in the colon for a longer period of time, meaning that more water is absorbed. This leads to harder stools and therefore increases the symptoms of constipation.